采取这种方式生长，平滑肌可分泌胶原，一种结构蛋白，是人体中除其它几种物质外组成血管壁的重要成分。
Grown this way, smooth muscle secretes collagen, a structural protein that is, among several other things in the body, an important component of the walls of blood vessels.
我至少花一小时来解释内皮细胞的保护作用、一氧化氮是我们血管壁的终极保护者。
I spend at least an hour defining the protective role of endothelial cells and nitric oxide functioning as the ultimate guardians of our blood vessels.
必备知识：总的来说，就是你的血液对血管壁和心脏压得过猛。
What you're supposed to know: Basically, your blood's pushing too hard on the sides of your blood vessels and your heart.
减少血管壁的过分扩张和损害的风险。
Reducing your risk of your vascular walls becoming overstretched and injured.
其中粘着在血管壁上的胆固醇是低脂密度脂蛋白，因此是‘坏’的。
The cholesterol which sticks to the vascular walls is called LDL and is therefore 'bad'.
当血管壁受损，胶原蛋白和组织因子暴露，循环血小板通过血管性血友病因子的作用发生黏着，并伴形状改变和ADP的释放。
When vessel walls are damaged, collagen and tissue factor are exposed, and circulating platelets adhere via von Willebrand factor and undergo a change in shape with the accompanying release of ADP.
因为还有第二种胆固醇，即低密度脂蛋白(LDL) LD L变得越来越小直到他们足够小能进入血管壁，然后依附在血管壁上。
Because the second kind, low-density lipoprotein (LDL), get smaller and smaller until they're tiny enough to enter the walls of your blood vessels and attach themselves like a bad houseguest.
在这张图的上边，血管壁细胞模仿毛细血管，，在另一边，模拟肺癌细胞的肺上皮细胞。
On one side of the sheet, blood-vessel cells mimic a capillary wall; on the other, lung-cancer cells mimic lung epithelial cells.
这些恶心的分子能够损伤动脉，从而使血管很难再维持血压正常并且会使胆固醇更容易附着在血管壁上。
These nasty molecules damage the arteries, making it harder for blood vessels to do their job of keeping blood pressure normal and making cholesterol more likely to stick like glue to artery walls.
由于人们认为提升高密度脂蛋白的水平有助于防止脂肪在动脉血管壁上沉积，因此在血液中增加高密度脂蛋白不失为消除低密度脂蛋白危害的好办法。
Because higher levels of HDL are thought to help prevent build-up of arterial plaque, boosting levels of it in the bloodstream could be a good way to fight harmfully high levels of LDL.
血管中脂肪斑块的堆积会导致动脉粥样硬化，而它则是由构成血管壁的细胞无法处理过多的脂肪造成的。
The build up of fatty plaques in blood vessels, which results in atherosclerosis, is a result of the inability of the cells lining the walls of these vessels to cope with too much fat.
马凡氏综合症要尽早发现，这至关重要，因为该病通常与主动脉的血管壁薄弱有关。
It's vital that Marfan's be diagnosed as soon as possible, as the condition is commonly associated with weakness of the wall of the aorta.
这种化学成分减少血管壁脂肪沉积，帮助预防动脉硬化保持血管健康。
The high levels of phytochemicals may help prevent hardening of the arteries by reducing plaque build-up and keeping blood vessels healthy.
事实上相当简单——血压是测量血液对血管壁产生的压力。
It's pretty simple — blood pressure is the measurement of the force of the blood against the walls of your blood vessels.
胆固醇是血液中的一部分，当血液开始粘附在血管壁上时，胆固醇也开始附着在血管壁上(怎么会没有'低密度脂蛋白血液'和'高密度脂蛋白血液'之分呢？)
Cholesterol is a part of blood and when blood starts sticking to the vascular walls so does the cholesterol in the blood (how come there's no 'LDL blood' and 'HDL blood'?)
研究主要集中在上皮细胞上，即那些分布在肠道和血管壁的细胞。
The research focused on epithelial cells, the cells that line the gut and blood vessels.
免疫系统对这些毒素的反应能够损害血管壁或使血凝块更轻易地形成。
The immune system's response to these toxins could harm vessel walls or make blood clot more easily.
其全过程中癌细胞突起直接与成血管细胞、血管内皮细胞及血管壁相连。
The cancer cells connected with angioblast cells, vascular endothelial cells and blood vessel walls in the whole process of angiogenesis.
其出血与血管内和血管周围改变血管壁的完整性，并且，它们亦在脂肪组织中聚集，并在此处释放化学物质引起炎症。
Their presence in and around blood vessels alters the integrity of vessel walls and they also collect in fat tissue, where they release chemicals that cause inflammation.
炎症期间，血管壁出现表面选择素能够特异性的粘附白细胞。
During inflammation, the vascular wall presents surface selections that adhere specifically to white blood cells.
接着，通过求解局部扩张血管段内的血液流动方程，导得血液流速、压力和血管壁切应力的分析表达式。
Through solving the blood flow equations for local vessel expansion region, the analytical expressions of blood flow velocity, pressure and wall shear stress were then obtained.
受伤以后，血小板粘在破损血管壁上，并且互相粘结，从而使蛋白质凝固，形成血栓。
After an injury, platelets stick to the walls of damaged vessels, to each other, and to clotting proteins, forming a plug.
使血管壁炎症限制在血管树特定解剖部位的分子机制还不明确。
Molecular mechanisms directing wall inflammation to restricted anatomic sites within the vascular tree are not understood.
在以上全过程中癌细胞突起直接与成血管细胞、血管内皮细胞及血管壁相连。
The cancer cells were connected with angioblast cells, vessel endothelial cell and blood vessel wall during the angiogenesis process.
在以上全过程中癌细胞突起直接与成血管细胞、血管内皮细胞及血管壁相连。
The cancer cells were connected with angioblast cells, vessel endothelial cell and blood vessel wall during the angiogenesis process.
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