当血管壁受损，胶原蛋白和组织因子暴露，循环血小板通过血管性血友病因子的作用发生黏着，并伴形状改变和ADP的释放。
When vessel walls are damaged, collagen and tissue factor are exposed, and circulating platelets adhere via von Willebrand factor and undergo a change in shape with the accompanying release of ADP.
近来，有关血管内皮生长因子家族对血液发生的作用受到广泛关注。
Recently the effect of vascular endothelial growth factor family on blood has been paid much attention.
RI被认为是胚胎发育、创伤愈合及肿瘤发生中新血管形成的一种调节因子。
Ri was recognized as a regulating element on formation of new blood vessels in embryo developing, wound healing, and tumor occurring.
机体炎症反应时释放的生物活性因子，不但可预示血管事件发生的危险性，而且还参与AS相关疾病的发病过程。
Biological active factors released in the inflammatory reaction not only indicate the as plaque stability, but play a significant role in as development.
结果表明，脑血流内皮舒张因子和内皮收缩因子之间的平衡紊乱是SAH后脑血管痉挛发生的机制之一。
It indicated that the imbalance of endothelium derived relaxing factor (NO) and the endothelium derived constricting factor (ET) was a major cause of vasospasm after SAH.
单因素分析显示，冠心病病史和糖尿病是NSTEMI患者6个月发生心血管事件的危险因子。
The monofactor analysis shows:the past history of angina and diabetes mellitus are the predicting risk factors of major cardiovascular event in the 6- month follow-up in NSTEMI patients.
血管内皮生长因子是目前所知唯一作用于血管内皮的生长因子，在血管发生中处枢纽地位。
Vascular endothelial growth factor is only one endothelium specific growth factor that plays a pivotal role in angiogenesis.
两组在骨折后3周和6周的骨形态发生蛋白2及血管内皮生长因子差异无显著性意义（P > 0.05）。
There was no significant difference of BMP-2 and VEGF between experimental group and control group at 3 weeks and 6 weeks after fracture (P > 0.05).
两组在骨折后3周和6周的骨形态发生蛋白2及血管内皮生长因子差异无显著性意义（P > 0.05）。
There was no significant difference of BMP-2 and VEGF between experimental group and control group at 3 weeks and 6 weeks after fracture (P > 0.05).
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