血管内皮细胞是连续被覆在全身血管内膜的一层细胞群，是具有保护血管作用的多酚在血管内活动的重要场所。
The endothelial cells which line our arteries are an important site of action for the vascular protective effects of polyphenols, " explains Corder ."
不过，过氧化氢也能提升血管内膜告诉周围肌肉舒张的电信号强度。
However, hydrogen peroxide can also act to increase the strength of electrical signals from the blood vessel's lining telling the surrounding muscle to relax.
她还说：“通常动脉血管内膜增厚是老龄化的标志；但对抗性强的年轻人动脉内膜也会增厚。”
Usually, thickening of the artery walls is a sign of age; however, young people with antagonistic traits already had thickening of the artery wall, she added.
目的：探讨兔髂动脉球囊剥脱术后局部灌注重组组织因子途径抑制物蛋白对血管内膜增生的抑制作用。
AIM to investigate the inhibitory effect of local perfusion of recombinant tissue factor pathway inhibitor protein on intima hyperplasia in atherosclerotic arteries after balloon stripping in rabbits.
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是术后血管再通的主要障碍。
The major complication of vascular recanalization is intimal hyperplasia which in due mainly to over-proliferation and migration of smooth muscle cells(SMC).
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
目的：探讨阿托伐他汀对大鼠颈总动脉内膜损伤后血管平滑肌细胞离子泵活性和血管内膜增生的影响。
Objective: to evaluate the influence of atorvastatin on ion pump activity of smooth muscle cells and intima proliferation in rats after arterial intimal injury.
目的研究阿托伐他汀对自发性高血压大鼠颈动脉外膜去除后血管内膜增生的影响。
Aim to investigate the effect of atorvastatin on intimal hyperplasia of carotid artery after adventitia removal in spontaneously hypertensive rats (SHR).
增殖细胞核抗原的表达与血管内膜增殖反应基本一致。
The expression of proliferative cell nuclear antigen showed the similar trend to the proliferative reaction of vascular intima.
颈动脉内膜-中膜厚度(IMT)是指颈动脉血管内膜和中膜之间的垂直距离。
The carotid intima-media thickness (IMT) refers to the vertical dimension between carotid intima and media.
目的：应用彩色多普勒超声观察糖尿病患者颈动脉内膜中膜厚度(IMT)并探讨其血管内皮功能及两者的相关性。
Objective: To probe the correlation between the carotid intima media thickness (IMT) and endothelial function in patients with diabetes by using Color Doppler Ultrasound.
目的：探讨超声彩色多普勒检测高血压及高血压伴糖尿病患者颈动脉早期动脉硬化血管内膜结构和血流动力学改变的意义。
Objective: To investigate the value of color Doppler ultrasound in detecting early atherosclerosis (carotid MT) and the hemodynamic changes of carotid artery in hypertension patients and diabetes.
目的探讨血管内膜肉瘤的临床及病理特点。
Purpose to explore the clinical features and histopathological character of intimal sarcoma.
模型组大鼠的大血管内膜损伤明显，平滑肌细胞增生，其中老年模型组大鼠的血管损伤较青年模型程度严重。
Rats in model group had obvious great vessel histopathology and proliferated SMC, while vessel injury in old model group were more serious than that of young model group.
目的：探讨稳态层流下的内皮细胞特征变化规律和血流切应力改变对血管内膜结构的影响。
Objective: to study the regular rule of changes in endothelial cells morphometric feature under steady flow state, and to investigate the effects of shear stress on the structure of vascular intima.
胸片与CT无法显示早期的气管内膜病变，对血管性病变的诊断价值有限。
Plain chest radiograph and chest CT were insensitive to early bronchial mucosal abnormalities. CT was limited in diagnosis of vascular disease.
同时，烟碱还有促进动脉粥样硬化和促进血管内膜增殖的作用，因此有促进再狭窄的作用。
Nicotine can also accelerate atherosclerosis and intimal proliferation, so may contribute to the development of restenosis.
当King于2002年初回到罗彻斯特大学后，他开始研究血细胞粘附血管壁的过程，血管壁即血管内膜层。
When King came to the University of Rochester in early 2002, he started studying the adhesion of blood cells to the vascular wall, the inner lining of the blood vessels.
结论：局部转染vegf165基因可抑制血管新生内膜增生及血管再狭窄，为将来血管内膜增生的基因治疗奠定基础。
CONCLUSION: Local transfection of VEGF165 gene restrains intimal hyperplasia and restenosis of vessels, which lays a foundation for future gene therapy of vascular intimal hyperplasia.
方法利用SD大鼠颈总动脉球囊损伤法造成颈动脉狭窄，观察颜氏益心方对颈动脉狭窄血管内膜厚度及管腔面积的影响。
Methods SD rat carotid artery stenosis was induced by balloon injury. The effects of Yan Yixin Fang on intimal thickness and lumen area of the narrowed carotid artery were observed.
辛伐他汀呈剂量依赖性抑制球囊损伤后血管内膜增生和多聚泛素、RC3的表达。
Simvastatin inhibited, in a dose-dependent manner, the intimal proliferation and the expression of polyubiquitin and RC3 mRNA and polyubiquitin protein.
应用血管内膜增生抑制剂后，内膜增生受到抑制，细胞增生性衰老的现象也趋于减少。
When we use all-trans retinoic acid, neointima formation is inhibited, and the phenomenon of replicative senescences is tend to decrease.
结果：严重肢体电击伤的血管尤其静脉血管血管壁肿胀、内膜粗糙、血管呈“串珠样改变”或血管内有泥沙样物质阻塞，血流速度减慢或血流通过。
Result: Blood vessels, especially veins wall were swelling, inner membrane became coarse, vessels had "beaded changes" or vessels were obstructed by sand - like materials. Blood flow lowered down.
血液流变学的变化是大黄抑制血管搭桥术后血管内膜增生的生理学基础。
Hemorheology change was the physiological basis of that inhibitory action to vascular intimal hyperplasia.
结论 血管内超声是评价支架内再狭窄的有效的手段， 支架内再狭窄患者有更大的斑块面积和内膜增生面积，而最小管腔面积变小。
Conclusion IVUS is the effective ways to access the ISR, the larger plaque area, neointimal area but smaller minimal lumen area were seen in patients with ISR.
目的研究动脉粥样硬化病变血管置入镍钛合金支架后血管内膜反应及卡托普利对此反应的影响。
Objective to study the intimal response after Nitinol alloy stenting in atherosclerotic lesion and the effect of captopril on the response.
MMP-2在粥样硬化冠脉斑块中有表达，表达的阳性部位为内膜的血管内皮细胞胞浆、巨噬细胞胞浆、平滑肌细胞胞浆和中膜的平滑肌细胞胞浆。
MMP-2 protein expression can be observed in atherosclerosis, mainly in macrophage, foam cells, endothelial cell, smooth muscle cell in endomembrane, smooth muscle cell and fibrocyre in tunica media.
MMP-2在粥样硬化冠脉斑块中有表达，表达的阳性部位为内膜的血管内皮细胞胞浆、巨噬细胞胞浆、平滑肌细胞胞浆和中膜的平滑肌细胞胞浆。
MMP-2 protein expression can be observed in atherosclerosis, mainly in macrophage, foam cells, endothelial cell, smooth muscle cell in endomembrane, smooth muscle cell and fibrocyre in tunica media.
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