血管内膜增生及水肿，并造成血管狭窄或阻塞。
The intima was hyperplastic and edematous and consequently the vessel lumen became narrower or was obstructed.
血液流变学的变化是大黄抑制血管搭桥术后血管内膜增生的生理学基础。
Hemorheology change was the physiological basis of that inhibitory action to vascular intimal hyperplasia.
目的研究阿托伐他汀对自发性高血压大鼠颈动脉外膜去除后血管内膜增生的影响。
Aim to investigate the effect of atorvastatin on intimal hyperplasia of carotid artery after adventitia removal in spontaneously hypertensive rats (SHR).
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是术后血管再通的主要障碍。
The major complication of vascular recanalization is intimal hyperplasia which in due mainly to over-proliferation and migration of smooth muscle cells(SMC).
辛伐他汀呈剂量依赖性抑制球囊损伤后血管内膜增生和多聚泛素、RC3的表达。
Simvastatin inhibited, in a dose-dependent manner, the intimal proliferation and the expression of polyubiquitin and RC3 mRNA and polyubiquitin protein.
应用血管内膜增生抑制剂后，内膜增生受到抑制，细胞增生性衰老的现象也趋于减少。
When we use all-trans retinoic acid, neointima formation is inhibited, and the phenomenon of replicative senescences is tend to decrease.
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
结论：偏心搭桥能够显著增加远心端血流量，抑制搭桥术后远心端吻合口对应面的血管内膜增生。
The difference was statistically significant (P<0.01). Conclusions: The eccentric bypass method could inhibit intimal hyperplasia at the opposite site of distal stoma by enhancing blood flow volume.
目的：探讨兔髂动脉球囊剥脱术后局部灌注重组组织因子途径抑制物蛋白对血管内膜增生的抑制作用。
AIM to investigate the inhibitory effect of local perfusion of recombinant tissue factor pathway inhibitor protein on intima hyperplasia in atherosclerotic arteries after balloon stripping in rabbits.
目的：探讨阿托伐他汀对大鼠颈总动脉内膜损伤后血管平滑肌细胞离子泵活性和血管内膜增生的影响。
Objective: to evaluate the influence of atorvastatin on ion pump activity of smooth muscle cells and intima proliferation in rats after arterial intimal injury.
结论：局部转染vegf165基因可抑制血管新生内膜增生及血管再狭窄，为将来血管内膜增生的基因治疗奠定基础。
CONCLUSION: Local transfection of VEGF165 gene restrains intimal hyperplasia and restenosis of vessels, which lays a foundation for future gene therapy of vascular intimal hyperplasia.
模型组大鼠的大血管内膜损伤明显，平滑肌细胞增生，其中老年模型组大鼠的血管损伤较青年模型程度严重。
Rats in model group had obvious great vessel histopathology and proliferated SMC, while vessel injury in old model group were more serious than that of young model group.
以上提示，氟伐他汀能有效抑制血管内膜过度增生，可能与其抑制平滑肌细胞增殖并可促进细胞凋亡有关。
Conclusion Fluvastatin inhibit excessive thickening of vascular intima effectively, which may partly be related to its inhibiting the proliferation of VSMCs and promoting the apoptosis of cells.
结论 血管内超声是评价支架内再狭窄的有效的手段， 支架内再狭窄患者有更大的斑块面积和内膜增生面积，而最小管腔面积变小。
Conclusion IVUS is the effective ways to access the ISR, the larger plaque area, neointimal area but smaller minimal lumen area were seen in patients with ISR.
结论 血管内超声是评价支架内再狭窄的有效的手段， 支架内再狭窄患者有更大的斑块面积和内膜增生面积，而最小管腔面积变小。
Conclusion IVUS is the effective ways to access the ISR, the larger plaque area, neointimal area but smaller minimal lumen area were seen in patients with ISR.
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