测定外周血血清中丙二醛（MDA）的含量。
方法血管康袋泡茶给大鼠连续灌胃15d，观察大鼠肝组织血清中丙二醛(MDA)含量。
MethodsFeed mice of Xueguankang Tea for 15 days and observe the content of MDA in liver and serum.
结果：与结扎组相比较电针能明显降低血清中丙二醛（MDA）的含量，提高超氧化物歧化酶（SOD）的活性。
Results:Compare with the ligation group the SOD level of the group with acupuncture elevated at the same time the MDA decreased.
测定血清中丙二醛（MDA）和超氧化物歧化酶（SOD）、乳酸脱氢酶（LDH）、磷酸肌酸激酶（CK）的含量。
Measured the contents of malondialdehyde(MDA), lactate dehydrogenase(LDH), creatine kinase(CK) and the activity of superoxide dismutase(SOD) in serum.
可明显逆转创伤小鼠血清及淋巴组织中ve含量的降低，丙二醛(MDA)含量的升高以及淋巴细胞膜流动性的降低。
It could reverse obviously the decreased ve contents and increased malondialdehyde (MDA) contents in serum and lymph tissues, reducing lymphocyte's plasma membrane fluidity in traumatized mice.
方法用丙二醛(MDA)和一氧化氮(NO)试剂盒测定不同组别大鼠中度脑损伤后脑组织及血清中MDA和NO的含量。
Methods the content of malondialdehyde (MDA) and NO in the brain tissue and serum of rats in different group was examined.
测定血清丙二醛含量和总SOD活性以及肝组织匀浆中的脂褐素含量。
The content of MDA in serum and lipofuscin in liver homogenate and the activity of T SOD in serum were detected.
目的通过对胎膜早破孕妇血清氧化应激指标—超氧化物歧化酶、丙二醛及维生素e的测定，分析3个指标在胎膜早破发病机制中的意义。
Objective to determine levels of maternal serum SOD, MDA and ve in patients with premature rupture of membranes (PROM) in order to analyze their significance in pathogenesis of PROM.
目的通过对胎膜早破孕妇血清氧化应激指标—超氧化物歧化酶、丙二醛及维生素e的测定，分析3个指标在胎膜早破发病机制中的意义。
Objective to determine levels of maternal serum SOD, MDA and ve in patients with premature rupture of membranes (PROM) in order to analyze their significance in pathogenesis of PROM.
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