循环中的LPA主要由激活的血小板释放，并引起血小板的聚集。
Activated platelets are the primary source of LPA in circulation, which in turn stimulate platelet aggregation.
血小板释放的一种酶，能在血液开始凝结时将凝血素转化为凝血酶。
A plasma protein that is converted into thrombin during blood clotting.
与RGDS显著的抗聚集效应相比，其对血小板释放反应的影响比较小；
The effects of RGDS on platelet release reaction were much less than those on platelet aggregation in same concentrations.
结论：从诱导血小板释放的角度，SJAMP是比adp更弱的一种血小板诱聚剂。
Conclusion: SJAMP was a weaker agonist that ADP in terms of platelets release reaction.
二磷酸腺苷（ADP）诱导血小板释放三磷酸腺苷（ATP）的量高于3月龄组，低于对照组；
Platelets of the experimental group released less ATP in response to ADP stimulation comparing with the control.
在外渗过程中，虫卵最初是如何粘附并穿透内皮，目前尚未阐明，虽然虫卵引发血小板释放的因子可能参与了这一过程。
It is unclear how eggs initially attach to the endothelium and initiate penetration during extravasation, although factors that are released from platelets in response to the eggs seem to be involved.
当血管壁受损，胶原蛋白和组织因子暴露，循环血小板通过血管性血友病因子的作用发生黏着，并伴形状改变和ADP的释放。
When vessel walls are damaged, collagen and tissue factor are exposed, and circulating platelets adhere via von Willebrand factor and undergo a change in shape with the accompanying release of ADP.
在初步形成的血小板阻塞释放的ADP刺激下，血小板发生聚集。
Local platelet aggregation is stimulated by ADP with the ultimate formation of the primary platelet plug.
脾切除术还消除了抑制骨髓生产或释放血小板的作用。
The splenectomy also removes a suppressant of bone marrow platelet production or release.
结论偏头痛的发作与血小板激活和释放有关，给予针对性的预防和治疗具有重要意义。
Conclusion the attack of migraine has something connected with the activation and the release of the platelet, so it is important to give therapy and prevention.
除了炎症反应外，血小板通过释放生长因子和趋化因子刺激修复得以募集循环中血管内皮组细胞。
In addition to inflammation, platelets stimulate repair by releasing growth factors and chemokines to recruit circulating vascular progenitor cells.
糖尿病高血压和血脂异常促进血小板内源性5 -HT释放并起协同作用。
Hypertension and dyslipidemia in coordination with each other enhanced the platelet 5-ht release in diabetes.
血小板具有黏附、聚集和释放反应的生理特性。
Physiologic characteristic of platelet includes adhesion, aggregation and release.
认为抗脑血栓注射液的抗血栓作用是通过降低全血粘度和TXB2浓度，从而抑制血小板的聚集和释放反应的结果。
From this trial, it was indicated that acti's reducing thrombosis action was the result of inhibiting aggregation and releasing of platelets through reducing whole blood viscosities and TXB2 level.
目的：研究MK- 447对胶原、ADP及血栓素a_2稳定类似物(STA_2)诱导的血小板变形、聚集和释放反应的影响。
AIM: To investigate the effects of MK-447 on platelet shape change, aggregation, and ATP release by collagen (col), ADP, and stable analogue of thromboxane A2 (STA2) in rabbits.
这些结果提示TMVA诱导的血小板聚集不依赖于释放反应和血栓烷a_2的形成。
Thus the platelet aggregation induced by TMVA was independent on the release reaction and the formation of TXA2.
结果糖尿病患者血小板聚集率、ATP释放均较健康人增高（P<0 .0 5） ；
Results Compared with healthy individuals, platelet aggregation and ATP release were increased in diabetes (P<0.05).
当血小板被激活时它会释放颗粒以促进血小板凝固并改变其形状使血小板凝固起来阻塞血管。
When activated, platelets release their granules to promote coagulation, change shape, and become sticky to aggregate and plug small vascular holes.
APT102是一种十分有趣的药物，它可以减少一种叫做ADP的物质，这种物质是由肿瘤细胞释放出来的，并可以促进血小板的生成。
APT102 is an especially interesting drug because it gets rid of a compound called ADP, which tumor cells release and which stimulates platelets to clump.
FDA明确了经过微生物检测系统释放检验后，用于常规储存和病人输血的单采血小板保存时间可增加为七天。
FDA clears apheresis platelets collected with certain systems for routine storage and patient transfusion up to 7 days when tested with a microbial detection system release test.
利用荧光血小板聚集仪测定了血小板ATP的释放量；
The release of ATP was measured by platelet fluorescent aggregometer.
还可以抑制血小板活性因子从而减少血凝，稀释血管及微血管，并阻碍巨噬细胞释放趋化及致炎介质。
They also inhibit platelet activation factor and thereby reduce thrombosis, dilate arteries and capillaries, and block the release of chemotactic and inflammatory mediators from phagocytes.
方法比浊法测定大鼠血小板聚集活性、荧光光度法测定血小板5 -HT释放反应、分光光度法测定血浆NO含量、血液流变全自动分析仪检测血液流变性。
Methods Platelet aggregation was quantified by turbidimetry, release of 5 HT was assayed by fluorescence technique, and content of NO in plasma was determined by spectrophotometry.
结论：芦丁可抑制PAF诱导的血小板聚集、HT释放及血小板内游离钙浓度增加。
Conclusion: Rutin could inhibit PAF induced platelet aggregation, 5 HT release and the increase of intraplatelet free calcium.
血小板激活后，二磷酸腺苷不仅从细胞类释放出来，而且可以激活血小板再活化，扩增整个血栓形成的过程。
After platelet activation, ADP is not only released from its intracellular storage granules but also further activates platelets, amplifying this process.
并释放出也血小板膜相关的纤维蛋白原、V和VIII因子、钙离子和血小板磷脂(PF - 3)。
Fibrinogen, factors V and VIII, calcium ions, platelet phospholipid (PF-3), associated with the platelet membrane are also released.
并释放出也血小板膜相关的纤维蛋白原、V和VIII因子、钙离子和血小板磷脂(PF - 3)。
Fibrinogen, factors V and VIII, calcium ions, platelet phospholipid (PF-3), associated with the platelet membrane are also released.
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