EGFR蛋白过表达和EGFR拷贝数改变的肿瘤内异质性无统计学意义。
Intratumoral heterogeneity of EGFR protein overexpression and EGFR copy number alteration was not significant.
除不良生存外，GLI1蛋白过表达与肿瘤分期及分析乳腺癌的淋巴结状况相关。
As well as poor survival, overexpression of the GLI1 protein was associated with tumor stage and lymph node status of the breast tumors analyzed.
EBL的发生与突变的P53蛋白过表达有重要关系，携带P53突变株的B淋巴细胞可能是形成肿瘤的靶细胞。
The positive cells expression of P53 had an important relation to the appearance of EBL. B cells with P53 mutation strain might be a target cell to format tumors.
结论APE1蛋白表达与HC C恶性表型有关，APE1蛋白过表达及细胞质表达的出现可能成为HCC预后不良的指标之一。
Conclusion the expression of APE1 is associated with the malignant phenotype of HCC. Overexpression and cytoplasm localization of APE1 can be used as a poorly prognostic marker for HCC.
极光激酶A（AIK）是一种调节细胞周期的丝氨酸/苏氨酸蛋白激酶，在多种肿瘤细胞系中过表达（1-3）。
Aurora A (AIK) is a cell cycle-regulated serine/threonine protein kinase that is overexpressed in many tumor cell lines (1-3).
细胞周期蛋白d1在癌症中经常过表达，可引起其过表达的因素很多，包括增加转录，翻译和蛋白质的稳定性。
Cyclin D1 is frequently over-expressed in cancers and its overexpression can be attributed to many factors including increased transcription, translation, and protein stability.
目的利用重组的胞内氯离子通道蛋白clIC1蛋白的过表达来了解该蛋白质在细胞中的定位。
Objective To investigate the localization of recombinant CLIC 1 protein in cultured cell line with its overexpression.
在肿瘤发生中，PIAS蛋白的过表达能抑制癌细胞的增殖并诱导其凋亡。
In tumor, overexpression of PIAS can inhibit the proliferation of cancer cells and induce cell apoptosis.
在肿瘤发生中，PIAS蛋白的过表达能抑制癌细胞的增殖并诱导其凋亡。
In tumor, overexpression of PIAS can inhibit the proliferation of cancer cells and induce cell apoptosis.
应用推荐