具体地，本发明显示E2-EPF5活性的抑制降低VEGF以及其他在低氧应答中由转录因子HIF-1调节的蛋白质的产生。
In particular, inhibition of E2-EPF5 activity is shown to reduce the production VEGF, as well as other proteins regulated the transcription factor HIF-1, in response to hypoxia.
具体地，本发明显示E2-EPF5活性的抑制降低VEGF以及其他在低氧应答中由转录因子HIF-1调节的蛋白质的产生。
In particular, inhibition of E2-EPF5 activity is shown to reduce the production VEGF, as well as other proteins regulated the transcription factor HIF-1, in response to hypoxia.
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