结论：苯代谢产物诱导的造血细胞凋亡和坏死可能是苯中毒导致的造血功能障碍的主要机制之一。
Conclusion Metabolites of benzene can induced hematopoietic cell apoptosis and necrosis, and give rise to aplastic anemia.
以内科急救原则对症治疗，可给予葡萄糖醛酸以增加苯的代谢产物的排出。
Symptomatic treatment to the principles of medical emergency may be given glucuronic acid to increase the excretion of benzene metabolites.
同时用HPLC检测反应体系中的苯并（a）芘含量、苯并（a）芘代谢物及DNA加合物酸水解后的产物。
The measurement of BaP, BaP metabolite and acid-hydrolysis products of DNA adducts by HPLC indicated that there were BPDE-DNA adducts.
气相色谱-质谱联用分析结果表明，DBT代谢产物中同时存在2 -HBP和二苯并噻吩砜(DBTO2)。
GC - MS analysis indicates that 2 - HBP and dibenzothiophene sulfone (DBTO2) simultaneously exist in the metabolite.
气相色谱-质谱联用分析结果表明，DBT代谢产物中同时存在2 -HBP和二苯并噻吩砜(DBTO2)。
GC - MS analysis indicates that 2 - HBP and dibenzothiophene sulfone (DBTO2) simultaneously exist in the metabolite.
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