因此，胰岛素抵抗是2型糖尿病致动脉粥样硬化的潜在病因。
Therefore, insulin resistance is believed to constitute the underlying cause of atherosclerosis in type 2 diabetes.
目的克隆、分离血管内皮细胞在致动脉粥样硬化因素作用下差异表达的基因，了解动脉粥样硬化发生的分子机制。
Objective to isolate and clone the differential expressed genes induced by atherogenic factors on vascular endothelium and to understand the molecular mechanism of atherosclerosis.
LDL受体介导的低密度脂蛋白胆固醇水平的升高是致动脉性粥样硬化的主要原因，与冠心病危险性的增加密切相关。
The LDL receptor defects cause the elevation LDLcholesterol in plasma. This is the main reason for atherosclerosis and is closely related to the risk of coronary heart disease (CHD).
最近几年，大量的基础和临床研究都证实低的或者很高的内皮剪切力加速改变一个致动脉粥样硬化内皮基因型。
In recent years a number of basic and clinical studies have provided evidence that low or turbulent wall shear stress promotes an atherogenic endothelial gene profile.
有许多因素影响血管剩留风险的存在，其中最为重要的是致动脉粥样硬化性血脂异常。
There are many factors responsible for residual risks of vascular events, atherogenic dyslipidemia is the most important factor.
目的：研究血管内皮细胞在致动脉粥样硬化因子作用下，差异表达基因的结构与功能。
Objective: to study the differences of expressed genes in vascular endothelial cells with the action of atherogenic factors and to explore the structure and function of these genes.
文章就人巨细胞病毒致动脉粥样硬化的依据和机制做了综述。
This article reviews the evidence and mechanism that human cytomegalovirus causes atherosclerosis.
我们假设多药抗药性相关蛋白-1介导的白三烯C4内位-外位转运是致血管系统动脉粥样硬化的重要机制。
We hypothesize that inside-outside transport of leukotriene C4 (LTC4) via MRP1 is a substantial proatherogenic mechanism in the vasculature.
我们假设多药抗药性相关蛋白-1介导的白三烯C4内位-外位转运是致血管系统动脉粥样硬化的重要机制。
We hypothesize that inside-outside transport of leukotriene C4 (LTC4) via MRP1 is a substantial proatherogenic mechanism in the vasculature.
应用推荐