其它改变，伴桥脑梗塞5例，小脑梗塞3例，大脑半球及底节梗塞8例，额顶叶脑白质脱髓鞘病变6例。
Others, Pons infarction in 5 cases, cerebellar infarction in 3 cases, cerebral hemisphere or base ganglion infarction in 8 cases, and leukoencephalopathy in 6 cases.
结论环己酮草酰二腙可诱导大鼠脑白质脱髓鞘、空泡样变，呈时间剂量相关性，随着时间延长和累积剂量增加，细胞凋亡及空泡形成增多；
It was related to the dosage and time. Along with the time extension and the cuprizone dosage increasement, the cell apoptosis increased and the demyelination aggravated.
PMD中不会有异常的强化，相反其他的脱髓鞘病变如肾上腺脑白质营养不良症其外周活动性的脱髓鞘病变可以强化。
Abnormal enhancement is not seen in PMD as opposed to some demyelinating disorders such as adrenal leukodystrophy in which enhancement is seen at the periphery of the active demyelination.
PMD中不会有异常的强化，相反其他的脱髓鞘病变如肾上腺脑白质营养不良症其外周活动性的脱髓鞘病变可以强化。
Abnormal enhancement is not seen in PMD as opposed to some demyelinating disorders such as adrenal leukodystrophy in which enhancement is seen at the periphery of the active demyelination.
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