最近，丙二酸辅酶a脱羧酶(MCD)已被确定为一个关键酶参与了心脏日常的脂肪酸氧化。
Recently, malonyl CoA decarboxylase (MCD) has been identified as a key enzyme involved in the regulation of cardiac fatty acid oxidation.
丙二酸辅酶A脱羧酶（MCD ）抑制剂类似的有益效果还体现在受需求诱导的猪在体心脏方面。
Similar beneficial effects of MCD inhibition are seen in the in vivo pig heart subjected to a demand-induced ischemia.
因此，丙二酸辅酶a脱羧酶(MCD)抑制剂代表着一个令人振奋的治疗缺血性心脏病的新靶点。
Hence, inhibition of MCD represents an exciting new target for the treatment of ischemic heart disease.
在大鼠心脏进行丙二酸辅酶a脱羧酶(MCD)基因筛除，同样可以使心肌缺血患者的心脏功能得到改进和恢复。
Genetic deletion of MCD from mice hearts also results in an improved recovery of heart function following ischemia.
在大鼠心脏进行丙二酸辅酶a脱羧酶(MCD)基因筛除，同样可以使心肌缺血患者的心脏功能得到改进和恢复。
Genetic deletion of MCD from mice hearts also results in an improved recovery of heart function following ischemia.
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