手术切除及组织病理学检查显示血管内皮损伤、脑组织坏死。
Resections and histopathologic observations of lesions showed damage of the endothelium of vessels and ischemia and cerebral necrosis.
脑梗死继发癫痫的发生在很大程度上决定于脑梗死的部位和脑组织坏死灶的多少。
Whether cerebral infarction will be seconded by epilepsy is determined by location and amount of infarction focus to great extent.
结果：局限性脑组织坏死为BCNU的血管毒性和神经毒性造成，与给药途径有关。
Results: The focal cerebral necrosis resulted from the BCNU toxicity of the blood vessels and cerebral nerves, and it is related to the way of infusion and drug quantities.
外科医生经常会在病人清醒的时候用电流刺激肿瘤周围的脑组织，观察病人的反应以避免切除那些没有坏死的部分。
Surgeons often electrically stimulate the area around the tumor while the patient is awake and can provide feeback, so they can avoid damaging critical tissue.
肉眼观：左上角的脑组织梗死表现为液化性坏死。最终坏死组织被清除后留下空洞。
Grossly, the cerebral infarction at the upper left here demonstrates liquefactive necrosis. Eventually, the removal of the dead tissue leaves behind a cavity.
研究了不同部位脑组织的病理变化，主要改变为大脑皮层神经细胞呈局部缺血性改变，少数皮层下有软化坏死灶。
Pathological examination of the brain of the rabbits revealed that there was local ischemic change of the neurons of the cortex and necrosis in the subcortical region in a few of the rabbits.
高倍镜下，在右侧的脑组织液化性坏死灶中许多巨噬细胞清除了坏死的细胞碎片。
At high magnification, liquefactive necrosis of the brain demonstrates many macrophages at the right which are cleaning up the necrotic cellular debris.
目的：探讨幼龄鼠未成熟脑组织对惊厥性脑损伤中细胞凋亡与坏死病理过程具有主动保护性抑制机制的分子生物学机理。
Objective: to explore the molecular biological base of internal protective response in premature brain which reduces the process of apoptosis and necrosis of neurons.
左上角的脑组织梗死表现为液化性坏死。
Grossly, the cerebral infarction at the upper left here demonstrates liquefactive necrosis.
左上角的脑组织梗死表现为液化性坏死。
Grossly, the cerebral infarction at the upper left here demonstrates liquefactive necrosis.
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