观察脑梗死体积、神经功能缺损评分和病理改变。
局灶脑缺血40分钟后再灌注时进行局部亚低温能减轻脑梗死体积。
Local mild hypothermia could decrease the volume of cerebral infarct, reperfusion following local ischemia for 40 minutes.
结论缺血性脑卒中患者急性期血小板活化增强，且与脑梗死体积相关。
Conclusion the activation function of platelet in the acute phase of patients with ischemic stroke are increased, and it is associated with the volume of cerebral infarction.
西比灵组脑梗死体积比明显小于缺血再灌注组（P<0 .0 5 ）。
The ratio of infarct volume of group using sibelium was smaller than that of ischemic reperfusion group( P< 0 05).
血清MMP-2和CRP与脑梗死体积、患者神经功能缺损程度呈正相关。
There were obvious positive correlations of MMP-2 and CRP levels to volume of infarct and degree of neurofunction impairment.
运用神经功能缺损评分和焦油紫染色观察动物的神经功能症状与脑梗死体积；
Neurological deficit score and infarct volume in all groups were compared to estimate ischemic injury.
目的研究先兆tia对其后脑梗死体积和临床严重度的影响，探讨TIA的临床意义。
Objective To explore the effect of prodromal transient ischemia attack (TIA) on infarction volume, clinical severity and prognosis in patients with subsequent cerebral infarction.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
黄体酮；脑梗死；白介素6；梗死体积。
结论：脑梗死急性期血清mmp - 9水平可能与梗死体积和近期预后密切相关。
Conclusion: Serum MMP-9 levels may be closely associated with the infarction volumes and the recent prognosis in acute cerebral infarction.
HE染色及免疫组化检测脑梗死灶体积、病理改变及CD34阳性细胞浸润情况。
The technique of TTC and HE staining and immunohistochemistry were used to detect the volume of infarction and pathological change and the infiltration of CD34 positive cells, respectively.
目的：研究脑梗死大鼠在不同电刺激治疗后血管构筑及梗死体积的变化。
AIM: To investigate blood vessel build and infarcted area of rats after stroke by different electric stimulation.
目的：研究脑梗死大鼠在不同电刺激治疗后血管构筑及梗死体积的变化。
AIM: To investigate blood vessel build and infarcted area of rats after stroke by different electric stimulation.
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