目的：探讨幼龄鼠未成熟脑组织对惊厥性脑损伤中细胞凋亡与坏死病理过程具有主动保护性抑制机制的分子生物学机理。
Objective: to explore the molecular biological base of internal protective response in premature brain which reduces the process of apoptosis and necrosis of neurons.
结论：缺血缺氧性脑病是引起新生儿惊厥的原因之一，窒息越重，EEG阳性率越高。
Conclusion: HIE is one of the main cause of neonatal convulsion. The more serious the asphyxia, the higher the positive rate of EEG.
获得性因素：由获得性因素引起脑的结构异常或代谢异常可产生致痫灶或降低惊厥阈值。
Acquired factors: factors from the acquired structural abnormalities of the brain or metabolic abnormalities can produce epileptic foci or reduce the seizure threshold.
结果：57例患儿中，病毒性脑炎、高热、低钙伴惊厥者无一例并发症发生，全部治愈出院； 缺血缺氧性脑病伴惊厥的患儿病情得到及时控制，好转出院。
Result:In the 57 cases of children with infantile convulsion, the concomitant by viral encephalitis, fever and hypocalcaemia in patients without complications occurred, and all cured.
结果：57例患儿中，病毒性脑炎、高热、低钙伴惊厥者无一例并发症发生，全部治愈出院； 缺血缺氧性脑病伴惊厥的患儿病情得到及时控制，好转出院。
Result:In the 57 cases of children with infantile convulsion, the concomitant by viral encephalitis, fever and hypocalcaemia in patients without complications occurred, and all cured.
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