结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的研究雌激素对雄性大鼠脑缺血再灌注损伤脑组织兴奋性氨基酸(EAA)含量的影响。
Objective To study the effects of estrogen on the contents of EAA in cortex of male rats during cerebral ischemia and reperfusion.
目的研究脑醒喷鼻剂对大鼠局灶性脑缺血再灌注损伤脑组织超微结构的影响。
Objective To study the effects of Naoxing Nasal Spray(NNS)on ultrastructure of brain tissues in rats with focal cerebral ischemia -reperfusion injury(CIRI ).
结论亚低温能减轻大鼠脑缺血再灌注损伤，降低脑组织HSP70及GFAP蛋白的表达。
Conclusion Mild hypothermia therapy may inhibit the expression of HSP70 and GFAP after cerebral ischemia and reperfusion injury in rats.
目的通过比较大鼠脑缺血再灌注损伤中基质金属蛋白酶- 9 (MMP - 9)的表达，研究中药制剂通心络对缺血脑组织的作用机制。
ObjectiveTo investigate the effect of Tongxinluo by the dynamic changes of the expression of matrix metalloproteinase-9 (MMP-9) on local cerebral ischemic-reperfusion injury in rats.
结论：插线法制作的缺血再灌注损伤模型可致小鼠学习记忆能力减退、脑单胺类神经递质含量降低。
Conclusions: Suture method ischemia reperfusion model can decrease the learning and memory ability, the monoamine neurotransmitter contents of brain in mouse.
目的：研究脑泰方提取物预处理减轻沙土鼠脑缺血再灌注损伤的作用及作用机理。
Objective To study the effects and mechanism of Naotaifang extract preconditioning on the cerebral ischemia-reperfusion injury in gerbil.
结论青年大鼠和老龄大鼠脑缺血再灌注脑组织损伤与NO含量降低和TNF增高有关。
Conclusions the brain ischemia reperfusion injury in young and the aged rats were correlated with the decrease of NO level and the increase of TNF.
目的观察脑缺血再灌注损伤时血清和脑组织内洋地黄素水平变化和内洋地黄素拮抗剂地高辛抗血清对脑缺血再灌注损伤的干预作用。
AIM To evaluate the changes of serum and brain tissue endoxin in model of bilateral cerebral hemisphere ischemic reperfusion injury, and effect of anti digoxin antiserum (an antagonist of endoxin).
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
结论补体及炎症反应参与脑组织缺血再灌注损伤。
Conclusions Complement and inflammatory reaction participated in cerebral ischemia-reperfusion injury.
目的：探讨脑泰方对缺血再灌注损伤沙鼠大脑皮层氨基酸类神经递质含量的影响。
Objective: Discuss the influence of Naotaifang on the content of amino acids neurotransmitter of cerebral cortex on cerebral ischemia reperfusion injury in Gerbils.
脑内注射AQP4基因后可升高大鼠脑缺血再灌注损伤早期脑内aqp4的表达水平。
Expression of AQP4 in the brain at the early period of cerebral ischemia reperfusion injury was increased after injection of APQ4 gene into the rat brain.
结论：针刺预处理脑组织提取液具有明显的抗脑缺血再灌注损伤作用。
Conclusion the brain tissue extract of rats after acupuncture preconditioning has the effect to resist the cerebral ischemia-reperfusion injury.
目的观察脑梗饮对大鼠局灶性脑缺血再灌注脑组织损伤的保护作用，并进一步探讨其作用机制。
Objective To measure the protective effects of Naogengyin on cerebral ischemia-reperfusion injury in rat and its mechanisms.
结论：脑泰方提取物预处理具有抑制沙土鼠脑缺血再灌注损伤的作用。
Conclusion: Naotaifang extract preconditioning inhibits the cerebral ischemia-reperfusions injury in gerbil.
结论达纳康可通过降低脑组织MDA和NO含量来预防缺血再灌注损伤。
Conclusion Tanakan may protect the CIRI by reducing the level of MDA and NO.
方法：通过兔全脑缺血-再灌注的病理模型，观察中药脑脉通对其损伤的保护作用。
Methods: the cerebral ischemia and reperfusion inJury model of rabbits was established to observe the protective effect of Naomaitong on them.
结论：MT对反复脑缺血再灌注损伤有显的保护作用，其机制可能与抑制脑水肿及抑制脑内升高的MDA、NO有关。
CONCLUSION: MT shows significantly protective effect against repeated ischemia reperfusion injury in mice, and the mechanism might be related to inhibit the rise of MDA and NO in brain tissue.
目的观察脑脊液生理生化成分变化，初步探讨肝缺血-再灌注损伤对脑的影响。
Objective To explore the effects of liver ischemia-reperfusion injury on the changes of physiology biochemistry in cerebrospinal fluid.
结论茶儿茶素可能通过降低脑细胞内钙离子浓度，保护脑组织对抗缺血再灌注损伤。
CONCLUSION It indicated that tea catechins had protective effects on the injury of cerebral ischemia and reperfusion in rats, and these effects may be related to calcium concentrations in brain.
本课题利用大鼠脑缺血再灌注模型，观察人参总皂甙对缺血再灌注后脑组织损伤的影响，目的是探讨人参总皂甙对大鼠缺血再灌注脑组织的保护作用机制。
In our study we investigated the influence of ginseng total saponin on brain tissue injury after ischemia-reperfusion by the model of brain ischemia-reperfusion with rat models.
NO可能参与了肾缺血-再灌注损伤对脑功能的影响。
NO may involve in the influence of the renal ischemia-reperfusion injury on the cerebral function.
NO可能参与了肾缺血-再灌注损伤对脑功能的影响。
NO may involve in the influence of the renal ischemia-reperfusion injury on the cerebral function.
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