仔细选择手术方法和加强术后治疗可以有效地避免脑再灌注综合症。
Cerebral reperfusion syndrome can be avoided by careful selection of the operation method and improved post-operative treatment.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的研究大鼠脑局灶性缺血再灌注模型中N甲基D天冬氨酸（NMDA）受体随时间的变化。
Objective To study the changes of N methyl D aspartate(NMDA) receptor in the rat brain during reperfusion after local ischemia.
目的：观察多塞平对急性脑缺血再灌注大鼠各脑区单胺类递质含量的影响。
AIM: To observe the effects of doxepin on monoamines transmitter in the rat brain after acute cerebral ischemia reperfusion.
目的：研究脑泰方提取物预处理减轻沙土鼠脑缺血再灌注损伤的作用及作用机理。
Objective To study the effects and mechanism of Naotaifang extract preconditioning on the cerebral ischemia-reperfusion injury in gerbil.
目的观察亚低温对颅内巨大动脉瘤夹闭手术中临时阻断动脉血流及缺血-再灌注期的脑保护作用。
Objective To observe the effect of mild hypothermia on cerebral protection during temporarily blocking partent arteries of intracranial giant aneurysms and ischemia-reperfusion period.
目的探讨大剂量甲基强的松龙(MP)对缺血再灌注大鼠脑保护作用的机制。
Objective to discuss the mechanism of large dose methylprednisolone (MP) to cerebral protection of rats after ischemic reperfusion.
目的：研究缺血再灌注对脑组织水肿和ATP酶活性的影响。
Objective: Study the influence on rat brain edema and ATPase activity with cerebral ischemia reperfusion.
结论：插线法制作的缺血再灌注损伤模型可致小鼠学习记忆能力减退、脑单胺类神经递质含量降低。
Conclusions: Suture method ischemia reperfusion model can decrease the learning and memory ability, the monoamine neurotransmitter contents of brain in mouse.
目的观察脑梗饮对大鼠局灶性脑缺血再灌注脑组织损伤的保护作用，并进一步探讨其作用机制。
Objective To measure the protective effects of Naogengyin on cerebral ischemia-reperfusion injury in rat and its mechanisms.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
目的测定脑缺血再灌注后小鼠脑内不同神经核团单胺递质及其代谢产物的动态变化。
AIM To determine the dynamic changes of monoamine neurotransmitters and their metabolites in various brain regions of cerebral ischemia reperfusion mice.
用兔全脑缺血模型，观察了缺血再灌注期脑组织钙积聚和超微结构改变。
Using the model of the rabbit brain ischemia the intracellular calcium accumulation and ultrastructure alteration of cerebral cortex were observed.
目的：探讨脑泰方对缺血再灌注损伤沙鼠大脑皮层氨基酸类神经递质含量的影响。
Objective: Discuss the influence of Naotaifang on the content of amino acids neurotransmitter of cerebral cortex on cerebral ischemia reperfusion injury in Gerbils.
结论：针刺预处理脑组织提取液具有明显的抗脑缺血再灌注损伤作用。
Conclusion the brain tissue extract of rats after acupuncture preconditioning has the effect to resist the cerebral ischemia-reperfusion injury.
目的研究雌激素对雄性大鼠脑缺血再灌注损伤脑组织兴奋性氨基酸(EAA)含量的影响。
Objective To study the effects of estrogen on the contents of EAA in cortex of male rats during cerebral ischemia and reperfusion.
结论补体及炎症反应参与脑组织缺血再灌注损伤。
Conclusions Complement and inflammatory reaction participated in cerebral ischemia-reperfusion injury.
目的：研究脑康胶囊对脑缺血再灌注家兔血液流变性的影响。
Objective: To investigate the influence of Naokang capsule on cerebral ischemia -reperfusion of rabbits in blood rheology.
结论亚低温能减轻大鼠脑缺血再灌注损伤，降低脑组织HSP70及GFAP蛋白的表达。
Conclusion Mild hypothermia therapy may inhibit the expression of HSP70 and GFAP after cerebral ischemia and reperfusion injury in rats.
目的通过比较大鼠脑缺血再灌注损伤中基质金属蛋白酶- 9 (MMP - 9)的表达，研究中药制剂通心络对缺血脑组织的作用机制。
ObjectiveTo investigate the effect of Tongxinluo by the dynamic changes of the expression of matrix metalloproteinase-9 (MMP-9) on local cerebral ischemic-reperfusion injury in rats.
目的观察犬心脏停搏(CA)复苏后再灌注期间脑矢状窦血浆内皮素- 1 (et - 1)及脑氧代谢变化。
Objective To study the changes of endothelin-1 (ET-1) concentration in sagittal (sinus) and cerebral oxygen metabolism after cardiac arrest (ca) and resuscitation in dogs.
缺血再灌注组脑皮层及海马CA1区苏木精-伊红染色示典型的迟发性神经元坏死。
The cells stained with haematoxylin and eosin displayed typical tardus neuron cellular necrosis in pallium and hippocampal CA1.
目的：探讨脑醒冲剂对大鼠脑组织缺血再灌注的保护作用及其可能的机制。
Objective: To explore the protective effect of Naox - ing Instant Granules (NIG) on reperfusion of cerebral ischemia (RCI) in rats.
目的研究脑醒喷鼻剂对大鼠局灶性脑缺血再灌注损伤脑组织超微结构的影响。
Objective To study the effects of Naoxing Nasal Spray(NNS)on ultrastructure of brain tissues in rats with focal cerebral ischemia -reperfusion injury(CIRI ).
目的观察缺血再灌注模型大鼠脑内神经生长因子的变化，探讨胡黄连对神经元的保护作用。
Objective To observe the expression of nerve growth factor in ischemic reperfusion rat brain and investigate its mechanism.
目的探讨疏血通对沙土鼠缺血再灌注脑组织的保护作用及其机制。
Objective to study the protective effect and mechanism of Shuxuetong on gerbil brain tissue in the area of ischemia reperfusion.
目的观察脑缺血再灌注损伤时血清和脑组织内洋地黄素水平变化和内洋地黄素拮抗剂地高辛抗血清对脑缺血再灌注损伤的干预作用。
AIM To evaluate the changes of serum and brain tissue endoxin in model of bilateral cerebral hemisphere ischemic reperfusion injury, and effect of anti digoxin antiserum (an antagonist of endoxin).
目的观察脑缺血再灌注损伤时血清和脑组织内洋地黄素水平变化和内洋地黄素拮抗剂地高辛抗血清对脑缺血再灌注损伤的干预作用。
AIM To evaluate the changes of serum and brain tissue endoxin in model of bilateral cerebral hemisphere ischemic reperfusion injury, and effect of anti digoxin antiserum (an antagonist of endoxin).
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