脊髓可塑性是脊髓损伤恢复的基础。
Spinal cord plasticity underlie recovery from spinal cord injury.
说明脑源性神经营养因子既参与脊髓的正常生理功能，也与其损伤后的可塑性有关。
Therefore, BDNF may be involved in the normal physiological function and the plasticity of spinal cord after damage.
目的：探讨大鼠脊髓损伤（SCI）后前肢功能训练引起可塑性变化的可能机制。
Objective:To explore the possible mechanisms of enhanced plasticity by forelimb training after spinal cord injury(SCI)in rats.
结论：单发佐剂性关节炎大鼠脊髓背角和脊髓灌流液中有关的神经肽CCK8、OFQ、EM2均发生了可塑性变化。
Conclusion: Plastic changes occurred in the spontaneous release and the content of dorsal spinal cord in CCK 8 ir, OFQ ir, EM 2 ir with adjuvant induced monoarthritis.
研究高位脊髓损伤慢性期心血管系统的可塑性变化，探讨发生自主神经反射异常的机理。
To explore the mechanism of autonomic dysreflexia by making investigation of the plastic change of cardiovascular system in the chronic stage of high level spinal cord injured rats.
目的：探讨实验性大脑皮层梗塞后同侧纹状体、丘脑及对侧脊髓前角的神经结构可塑性。
Objective: To observe the neural structural plasticity occurred in ipsilateral striatum, thalamus and contralateral anterior horn of spinal cord after experimental cerebral cortex infarction in rats.
脊髓损伤可激活自体室管膜细胞的原位增殖及迁移，后者具有一定的可塑性，参与伤后脊髓的结构修复。
The spinal cord injury stimulates the proliferation of ependymal cells in sim, the ependymal cells participates in the rehabilitation of spinal cord injury and represent neural plasticity.
脊髓损伤可激活自体室管膜细胞的原位增殖及迁移，后者具有一定的可塑性，参与伤后脊髓的结构修复。
The spinal cord injury stimulates the proliferation of ependymal cells in sim, the ependymal cells participates in the rehabilitation of spinal cord injury and represent neural plasticity.
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