结论：诱发电位时频参数比时域参数能更灵敏地反映大鼠脊髓压迫损伤。
Conclusion: The time-frequency parameters are more sensitive in monitoring spinal cord compressional injury in rat than those in time domain.
模型对照组、干细胞移植组大鼠采用重物压迫法建立脊髓损伤模型。
Spinal cord injury models were established by compression method in rats of model control group and stem cell transplantation group.
目的探讨X线照射能否促进大鼠压迫型脊髓损伤区结构及损伤后功能的恢复。
To explore whether X-irradiation can enhance the functional and structural recovery of the injured spinal cord of rats.
目的观察大鼠坐骨神经慢性压迫损伤后脊髓背角浅层神经元超微形态变化。
Objective To observe the ultrastructure changes of sensory neurons in the spinal dorsal horn after chronic constriction injury (CCI) of the sciatic nerve in rats.
目的观察慢性压迫性脊髓损伤后骨骼肌形态学改变及其成肌细胞的增殖动力学变化。
Objective To observe the morphological changes and myoblast proliferation dynamics of skeletal muscle after chronic spinal cord compression in rats.
结论：因前方压迫产生神经损伤，尤其对于仅由中柱的骨折移位而致脊髓损伤者，侧前方减压术是首选方法。
Conclusion: Anterolateral decompression is the first selected method when neurological deficit is generated from anterior compression, especially when merely the middle column is involved.
脊髓压迫性损伤是神经系统的常见病，其中颈髓压迫性损伤最常见。
The cervical spinal cord compression injury is a kind of common disorder in nervous system.
目的：了解脊髓硬膜外血肿(SEH)与压迫性脊髓损伤的关系。
Objective: to investigate the relation between spinal epidural hematoma (SEH) and compressive spinal cord injury.
创伤性高位截瘫是颈椎骨折、移位、骨片压迫损伤脊髓所致的最严重的颈髓损伤。
Traumatic high paraplegia is the most serious damage of cervical spinal cord caused by fracture, dislocation of cervical spine, or the sclerites press and injure the spinal cord.
结果通过对犬行为学、影像学、电生理及组织学观察，对犬颈脊髓慢性损伤符合颈脊髓慢性压迫症病理改变的特点。
Results Postoperative behavior, radiological examination and histological observation were coincidence of the character of pathologic change of chronic cord compression myelopathy.
结论脊髓损伤后持续性压迫使损伤脊髓血流量恢复减慢，病理损害加重。
Conclusion Continued compression aggravates pathologic injury through affecting the recovery of local spinal cord blood flow.
结论脊髓损伤后持续性压迫使损伤脊髓血流量恢复减慢，病理损害加重。
Conclusion Continued compression aggravates pathologic injury through affecting the recovery of local spinal cord blood flow.
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