甘油三酯脂肪酶基因家族成员包括LPL、HL和胰脂酶等，他们均参与脂蛋白的代谢，其中，LPL和HL专司甘油三酯和磷脂的水解，在血管内皮表面发挥作用。
They all involve into the metabolize of lipoprotein. LPL and HL are in charge of thehydrolyze of triglyceride and phospholipid on the surface of vessel endothelium.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
脂蛋白脂肪酶(LPL)是脂蛋白代谢的关键酶，LPL基因变异常可导致异常脂蛋白血症。
Lipoprotein lipase (LPL) is a key enzyme of lipid metabolism, its primary function is the hydrolysis of the core triglycerides of circulating chylomicrons and very low density lipoproteins.
脂蛋白脂肪酶(LPL)是脂蛋白代谢的关键酶，LPL基因变异常可导致异常脂蛋白血症。
Lipoprotein lipase (LPL) is a key enzyme of lipid metabolism, its primary function is the hydrolysis of the core triglycerides of circulating chylomicrons and very low density lipoproteins.
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