目的评价纤溶酶在治疗结核性胸膜炎胸膜肥厚中的效果。
Objective To evaluation the effect of plasmin in the treatment of Pleural Thickening by Tuberculous Pleurisy.
目的探讨结核性渗出性胸膜炎胸腔积液纤维蛋白原含量与胸膜肥厚、粘连的关系。
Objective To investigate the relationship of concentration of pleural effusion in tuberculous exudative pleurisy with pleural thickness and adhesion.
结论胸腔内注射尿激酶能减少胸腔分隔、多房的形成。减少胸膜肥厚，改善肺功能。
Conclusion Intrapleural urokinase not only reduces pleural adhesion, loculation and thickening of the pleura, but also improves lung function.
局限性胸膜肥厚粘连，无胸水及钙化征象，肿块与增厚的胸壁之间有条状低密度影。
There were localized pleural thickening adhesion and strip-like low density lesion between mass and thickened chest wall.
结论正确认识及测量正常肺尖及其沿肋骨腋中线内缘的伴影，有助于鉴别胸膜肥厚，对阅读胸片有较大帮助。
Conclusion Correctly understanding and measuring the apex pulmonis companion shadow, it 's good to differentiate from hypertrophic pleura or reading chest films.
结论胸膜腔内注入尿激酶，能有效溶解结核性包裹性胸腔积液中的纤维分隔，明显降低胸膜肥厚的程度和粘连发生的机会。
Conclusion the intrapleural urokinase injection can effectively dissolves the fiber partition in loculated effusions by tuberculous pleurisy and prevent pleural thickening and adhesion.
结论正规抗痨配合中医药治疗有助于松解肥厚粘连的胸膜，恢复肺的生理功能。
Conclusion The normal anti-consumption therapy combined with Chinese medicine is conducive to relieve pachynsis and adherence of tuberculous pleuritis and recover the lung function.
结论正规抗痨配合中医药治疗有助于松解肥厚粘连的胸膜，恢复肺的生理功能。
Conclusion The normal anti-consumption therapy combined with Chinese medicine is conducive to relieve pachynsis and adherence of tuberculous pleuritis and recover the lung function.
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