目的：研究丙泊酚对慢性神经痛大鼠脊髓星形胶质细胞激活的影响。
Objective: To investigate the influence of propofol on the activation of astrocytes in the spinal cord of rats with chronic neuropathic pain.
本发明的化合物具有显著的抗癌，抗氧化，消除自由基及抑制小胶质细胞激活的药理活性。
The compounds of the invention has substantial pharmacological activity of anticancer, oxidation resistance, free radical remove and repressing activation of small cells.
结论：GBE可能通过抑制小胶质细胞激活从而延缓EAE小鼠脱髓鞘进程，提示GBE对多发性硬化具有一定的治疗作用。
Conclusion: GBE can delay demyelination process in EAE mice by inhibiting microglial activation, suggesting that GBE has potential to treat multiple sclerosis in future.
结论：糖尿病是脑缺血再灌注损伤重要因素之一；内皮素-3和星形胶质细胞激活可能是糖尿病小鼠神经细胞损伤加重恶化的机制之一。
CONCLUSION: Diabetes is one of the important causes of cerebral ischemia reperfusion injury, and et 3 and activated astrocytes may worsen the neural injuries in diabetic mice.
这些细胞虽然不一定能直接合成和分泌胶原，但他们的激活，已成为胶质生成细胞的重要来源。
While these cells are not necessarily directly collagen synthetic and secreted, their activation has became an important source of colloid generated cells.
观察到DRG内神经内膜巨噬细胞和卫星神经胶质细胞被长期的激活，这些细胞的反应与疼痛关联行为具有很好的相关性。
Long-term activation of endoneurial macrophages and satellite glial cells in the DRG was observed, and the reactions of these cells correlated well with pain-related behavior.
而星形胶质细胞的激活支持和保护大脑免受脑缺血损伤。
The activation of astrocytes help ischemic brain repair and prevent from injury.
非活化的星形胶质细胞对PHA刺激活化的T细胞增殖具有明显的抑制作用，而活化的星形胶质细胞的抑制作用减弱。
In contrast to the weaker effect induced by the activated astrocytes, the non-activated astrocytes had a significant inhibitory effect on PHA-stimulated T cell proliferation.
目的：研究L5脊神经结扎模型大鼠脊髓星形胶质细胞的激活与痛行为之间的关系。
Objective: To investigate the relationship between the spinal astrocytic activation and neuropathic pain behavior in L5 spinal nerve ligation (SNL) rats.
目的：探讨海马区星形胶质细胞的激活与3硝基丙酸（3NPA）预处理诱导脑缺血耐受的关系。
AIM: To investigate the relationship between astroglial activation state and ischemic tolerance induced by low dose of 3 nitropropionic acid (3 NPA) in gerbil hippocampus.
研究人员发现，小鼠的星形胶质细胞可以产生化学增活素作为对TNFR激活以后的应答。
The researchers found that astrocytes in mice were producing chemokines in response to activation of their TNFR molecules.
研究人员发现，小鼠的星形胶质细胞可以产生化学增活素作为对TNFR激活以后的应答。
The researchers found that astrocytes in mice were producing chemokines in response to activation of their TNFR molecules.
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