结论：子前期胎儿中性粒细胞的激活与母体中性粒细胞的激活相关，胎儿循环中存在中性粒细胞的激活，推测中性粒细胞在子前期胎儿病率中有一定作用。
The activation of neutrophil in mother of preeclampsia is related to the activation of neutrophil in fetal circulation. Neutrophil could play a role in fetal morbidity.
随着胎儿的成长，母体的血液循环加重。
With the growth of fetal and maternal blood circulation to increase.
快速地确定母体气道、呼吸及循环的情况并且确保足够的气道供气能避免母体和胎儿缺氧。
Rapid assessment of the maternal airway, breathing, and circulation and ensuring an adequate airway avoids maternal and fetal hypoxia.
结论本研究为多普勒超声早期预测胎儿高动力循环状态提供了理论依据。
Conclusion This study offers a theoretic basis to the early prediction of fetal hyperdynamic circulation with Doppler ultrasound.
当她们暴露于污染的空气之中，污染物便会进入她们的血液并再透过胎盘及脐带进入胎儿的血液循环系统中，严重影响胎儿的成长及发育。
When exposed to ambient air pollution, pollutants will enter a pregnant woman's bloodstream and pollutants can then reach the foetus' circulation through the placenta and umbilical cord.
目的：探讨利用母血循环中胎儿细胞进行产前诊断的最佳采血时间。
Objective: To investigate the optimum time of maternal blood sampling for prenatal diagnosis with fetal cells.
在整个妊娠期间，胎儿的营养摄入主要是通过母子血液循环获得的。
During the whole of gestation period, the nutrition of fetus is ingested by the blood circulation between maternal and fetus.
相比之下，胎儿并没有非常经典的炎症反应。我们知道，母体循环中的细胞因子并不能透过胎盘，因此不能到达胎儿。
By contrast the fetus is not in a very classic inflammatory state and we know that the cytokines which are circulating in the mother cannot cross the placenta so they do not reach the fetus.
当粘连发生，胎盘内血液循环受阻将直接影响胎儿发育。
When placental adherence happens, blood circulation of villus in the placenta could be retard, and then the developing of fetus were affected.
结论：脐血和脐带血管adm和ET - 1表达改变可能与妊高征胎儿胎盘之间血液循环调控有关。
Conclusion The changes of ADM and ET-1 in umbilical cord plasma and vessel may related to regulation of fetoplacental circulation in PIH.
从这些变化与胎儿-胎盘循环的关系和对妊高征发病机理的意义进行了讨论。
The relation between the changes and the fetal-placental circulation, as well as the pathog…
从这些变化与胎儿-胎盘循环的关系和对妊高征发病机理的意义进行了讨论。
The relation between the changes and the fetal-placental circulation, as well as the pathog…
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