该病导致显著的胆汁淤积和肝内胆管增生、纤维化、肝硬化。
This leads to marked cholestasis with intrahepatic bile duct proliferation, fibrosis, and cirrhosis.
镜下见，肝外胆道闭锁导致肝脏大量黄绿色胆栓、假胆管增生(见于下面的中央)、广泛纤维化。
Microscopically, extrahepatic biliary atresia leads to this appearance in the liver, with numerous brown-green bile plugs, bile duct proliferation (seen at lower center), and extensive fibrosis.
肝病理组织学检查发现BDL组胆管增生，纤维结缔组织明显增生，肝细胞索排列紊乱，胀亡增加。
Hepatic histopathological examination showed proliferation of bile duct and fibrous connective tissue, obvious increase of hepatic cell oncosis and liver cell cord derangement in BDL group.
结缔组织中散在淋巴细胞也有小胆管的增生。
Within this collagenous tissue are scattered lymphocytes as well as a proliferation of bile ducts.
早期胆肠内引流术后，随着增生的胆管减少，胆管上皮细胞凋亡明显减少。
Significantly reduced apoptosis in biliary epithelial cell is accompanied by disappearance of bile duct hyperplasia after biliary drainage.
感染后期，肝脏质地变硬，胆管和结缔组织增生，胆汁淤积。
At late stage, the main changes were proliferation of bile canaliculi and connective tissue, cirrhosis and cholestasis.
结果胆管梗阻后，胆管黏膜上皮坏死脱落、纤维结缔组织增生、管壁增厚。
Results After bile duct obstruction, the bile duct wall presented with epithelial necrosis, fibrous tissue proliferation and thickening.
胆管修复术后胆管局部纤维组织增生程度、胶原含量与胆管梗阻时程呈正相关。
The extent of fibrous tissue proliferation after prosthesis and collagen content at the anastomosis site were positively correlated to biliary obstruction duration.
D组胆管壁组织呈慢性增生性炎症改变。
Tissue of bile duct wall in group D had chronic proliferation inflammation.
D组胆管壁组织呈慢性增生性炎症改变。
Tissue of bile duct wall in group D had chronic proliferation inflammation.
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