目的探讨榄香烯乳治疗胃癌细胞的作用机制。
Purpose To identify the mechanism of elemene on gastric cancer cell.
缺氧的环境是胃癌细胞增殖的关键因素之一。
Hypoxia microenvironment is one of the crucial factors of the gastric cell proliferation.
目的：探索红毛五加多糖对胃癌细胞的抑制作用。
Objectives:To investigate the inhibitory effect of Acanthopanax giraldii Harms Var Hispidus Hoo polysaccharides(AGP) on the gastric cancer cell.
目的探讨土槿乙酸对人胃癌细胞生长的影响及机制。
Objective To investigate the effect of pseudolaric acid B on growth of human gastric carcinoma cells in vitro.
目的：研究人参皂苷对胃癌细胞、大肠癌细胞的作用。
OBJECTIVE:To study the effect of ginsenoside to the gastrocolic cancer cells.
目的：探讨容量调控氯通道在人胃癌细胞增殖中的作用。
AIM: To study the role of volume regulated chloride channel in the proliferation of human gastric carcinoma cells.
本文应用流式细胞术对胃癌细胞的DNA含量进行了分析。
Using flow cytometry the DNA content of gastric cancer cells Were analysed .
现在，逆转人胃癌细胞的研究已成为生命科学研究前沿领域。
Now human gastric cancer reversion research has become leading edge field in life science research.
目的探讨TROP2基因在胃癌细胞迁移侵袭过程中的作用。
Objective To investigate the role of TROP2 in migration and invasion of human gastric cancer cells.
目的：探讨特异性微小rna抑制剂对胃癌细胞增殖的影响。
AIM: to investigate the effects of miRNA-specific inhibitors on proliferation of gastric cancer cells.
目的：研究泛素蛋白酶体抑制剂对胃癌细胞增殖和凋亡的影响。
AIM: to investigate effects of ubiquitin-proteasome inhibitor on proliferation and apoptosis of gastric carcinoma cells.
前言胃癌细胞的侵袭与转移是影响胃癌患者预后的最根本原因。
PrefaceThe invasion and metastasis of gastric cancer is the most essential reason that affect the prognosis of gastric cancer.
结果新疆紫草素能够抑制胃癌细胞的增殖，并呈浓度时间依赖性。
RESULTS the alkannin could inhibit the proliferation of the cells with dependence of concentration and time.
目的探讨CD 40配体激活对胃癌细胞生长、增殖和凋亡的影响。
Objective to observe the effects of CD40 ligand activations on the growth, proliferation and apoptosis of gastric cancer cell lines.
目的探讨锌带基因ZNRD1在多药耐药胃癌细胞中的表达和作用。
Objective To study the expression and function of zinc ribbon gene ZNRD1 in drug resistant cells of gastric cancer.
结果：该多糖对胃癌细胞的增殖和胃癌干细胞均有显著性抑制作用。
Results:AGP inhibited the proliferation of the gastric cancer cell and the stem cell of the gastric cancer.
结论：四环素调控系统基本上可以控制毒素基因在人胃癌细胞中的表达。
Conclusions: the chimeric gene can be controlled by tetracycline-controlled system and result in cellular toxicity in the absence of tetracycline.
结果：该多糖不但抑制胃癌细胞的增殖，且对其干细胞有显著抑制作用。
Results: AGP inhibited not only the cell's proliferation, but also the stem cell significantly.
结论LR可能通过影响药物蓄积和细胞凋亡参与对胃癌细胞MDR的调节。
Conclusion LR might take part in mediation of MDR in gastric cancer cells through interfering with drug accumulation and cell apoptosis.
因此，靶向脂质体可以把包裹的抗癌药物专一地送给胃癌细胞，井杀伤它们。
Thus, the targeted liposomes can specifically deliver the antitumor drugs to gastric cancer cells and kill them.
胃癌细胞RUNX3蛋白表达与淋巴转移和远处转移有关（P<0.05）。
RUNX3 expression was correlated with lymph node metastasis and distant metastasis (P<0.05).
结果巴豆生物碱能改变细胞形态，诱导人胃癌细胞SGC- 7901凋亡。
RESULT the Cotton Alkaloid could significantly influence the shape of SGC-7901 and induce the cell apoptosis.
结论免疫毒素LHRH - PE40对胃癌细胞的增殖状态有明显的抑制作用。
Conclusions LHRH-PE40 has obvious inhibition effect on the proliferation of gastric cell lines.
结论：E - CD减弱表达与胃癌细胞分化差、浸润型生长及预后较差密切相关。
Conclusion: the decreased expression of E-CD might be responsible for the cellular dedifferentiation, the infiltrative growth and poor prognosis of gastric carcinomas.
结果淋巴管内皮、血管内皮及部分（38%）胃癌细胞均可表达VEGFR-3蛋白。
Results VEGFR-3 were expressed in lymphatic endothelium, vascular endothelium and 38% of gastric cancer cells.
胃癌细胞诱导损伤间皮后，受损残余的间皮可以反作用于癌细胞，使其迁移转移力提高。
The injuryed mesothelial cells could up-regulate the abilitys of invasion and metastasis in gastric cancer cells.
通过倒置荧光相差显微镜、光镜、透射电镜等技术观察转染基因的胃癌细胞的形态学变化。
The change of morphology of the transfected cancer cells were observed under inverted fluorescent contrast phase microscope, light microscope and transmission electron microscope.
目的：研究肿瘤细胞分化诱导剂尿多酸肽(CDA - II)对人胃癌细胞的体外抑制作用。
Objective: to study the inhibitory effect of Uroacitide (CDA-II) on human gastric cancer cell in vitro.
目的：研究四环素调控系统对DT390-VEGF165融合基因在人胃癌细胞中表达的影响。
Objective: To study the influence of tetracycline-controlled system on expression of the DT390-VEGF165 fusion gene in human gastric carcinoma cell.
结论KDR基因启动子可调控双自杀基因系统选择性杀伤表达KDR胃癌细胞及血管内皮细胞。
Conclusions the double suicide gene driven by KDR promoter has specific killing effect on KDR-expressing gastric tumor cells and venous endothelial cells in vitro.
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