血管内皮细胞生长因子(VEGF)及其受体在肿瘤血管新生中起重要作用，阻断VEGF的生成已成为抗肿瘤治疗的新靶点。
Vascular endothelial growth factor (VEGF) and its receptor play a critical role in tumor-associated angiogenesis and have become the new targets of anti-tumor therapy.
新生血管大量形成在实体瘤的生长和转移中起着关键的作用。血管内皮生长因子(VEGF)是介导肿瘤血管生成的最主要因素。
New vessel formation plays a key role in tumor growth and transforming, and the Vascular endothelial growth factor (VEGF) is the most important factor inducing tumor vessel formation.
新生血管生成在肿瘤的发生和生长过程中是一个至关重要的事件。
Angiogenesis is one of the crucial events for cancer development and growth.
抑制肿瘤新生血管的生成有望降低甚或遏制对非小细胞肺癌化疗的耐药性。
Inhibition of angiogenesis in tumor is expected to reduce or inhibit drug resistance to NSCLC.
血管生成促进因子和血管生成抑制因子的平衡控制着肿瘤新生血管的形成。
Formation of tumor new vessels is controlled by balances between angiogenic stimulators and inhibitors.
组织研究显示使用联合治疗肿瘤生长更缓慢，更少侵犯邻近组织和在新生成的血管中浓度更低。
Tissue studies showed that, under the combination therapy, tumors grew more slowly and less invasively and showed a lower density of newly formed blood vessels.
内皮抑素是一种抗血管生成的抑制因子，它特异性地作用于新生微血管的内皮细胞，其水平与肿瘤血管生成有着明显的相关性。
Endostatin is a kind of angiogenesis inhibitor, specific effecting in the new microvascular endothelial cells. The level of endostatin and the angiogenesis of tumor have a close correlation.
肿瘤细胞释放可溶性的促血管生成因子诱导新生血管生成。
Tumor cells release soluble angiogenic factors inducing neovascularization, a process referred to as the "angiogenic switch".
然而，作为对血管生成蛋白释放的反应，孕期或机体发生创伤、肿瘤等时处于静止状态的毛细血管新生过程被触发而发生增殖。
However, in response to angiogenic proteins released during pregnancy, wound healing, and tumor growth, capillaries in this quiescent vasculature can be triggered to proliferate.
肿瘤的生长与新生血管的形成和血流量增加有关，血管生成在肿瘤的生长、侵袭与转移过程中起重要的作用。
Tumor growth is associated with neovascularization and the increase of blood flow, and angiogenesis plays a key role in tumor growth, invasion, and metastasis.
有证据显示，免疫抑制、新生血管生成及外科手术等因素可使肿瘤休眠细胞激活，从而引发肿瘤的复发和转移。
Evidence has shown that immunological suppression, angiogenesis in tumor tissues as well as surgery may all play roles in tumor dormancy activation, and thus lead to tumor reoccurrence and metastasis.
采用新生血管计数实验检测UE-1对肿瘤组织血管生成的影响；
The inhibition of UE-1 on chick chorioallantoic membrane (CAM) angiogenesis was studied by CAM assay.
当血管生成开关打开，肿瘤内有足够的新生血管长入，提供营养并带走代谢产物，肿瘤便获得了进一步生长、转移的能力。
When tumor grows to certain extent, new vasculature was needed to access to provide nutrition and carry away the waste.
当血管生成开关打开，肿瘤内有足够的新生血管长入，提供营养并带走代谢产物，肿瘤便获得了进一步生长、转移的能力。
When tumor grows to certain extent, new vasculature was needed to access to provide nutrition and carry away the waste.
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