方法采用两肾一夹型肾血管性高血压大鼠模型。
METHODS The renovascular hypertension model was induced in rats by two-kidney-one-clip method.
方法：建立二肾一夹（2K1C）肾血管性高血压大鼠模型。
Methods Two kidney 1 clip (2K1C) rats were used to reestablish the model of renovascular hypertension.
目的：研究肾血管性高血压大鼠运动区NPY阳性神经元的变化。
Purpose: to research the changes of NPY neurons in the brain motor area in renovascular hypertensive rats.
目的研究葡萄糖、胰岛素对肾血管性高血压大鼠脑梗死的不同影响。
Objective To study the different effects of glucose and insulin on cerebral infarction of renal vascular hypertension in rats.
目的探讨黄芪注射液对肾血管性高血压大鼠左心室肥厚及心肌纤维化有无逆转作用。
Objective To explore whether the Astragalus injection (AI) has effect for reversing left ventricular hypertrophy and myocardial fibrosis induced by renal vascular hypertension in rats.
目的动态观察易卒中型肾血管性高血压大鼠(RHRSP)血液中凝血系统和纤溶系统的活性改变。
Objective To study the activity changes of coagulation and fibrinolysis in stroke prone renovascular hypertensive rats (RHRSP).
目的探讨一氧化氮前体L-精氨酸对肾血管性高血压大鼠心血管组织局部肾素-血管紧张素系统（RAS）的影响。
Objective To investigate the role of L-arginine on renin-angiotensin system(RAS) in cardiovascular tissue of rats with renovascular hypertension.
方法在双肾双夹法复制肾血管性高血压模型的基础上，以胶原酶加肝素脑内注射诱发脑出血，建立高血压性脑出血大鼠模型。
Methods On the base of renovascular hypertensive rats (RHR) model, the hypertensive cerebral hemorrhage rat model was induced by injecting collagenase and heparin into the brain.
实验结果表明粉防己碱可部分逆转肾血管性高血压所致的大鼠左心室肥厚，降低心肌胶原含量，升高肌球蛋白ATP酶活性。
The results suggest that Tet or Ena partially reduce the hydroxyproline content and elevate myosin ATPase activity of hypertrophied myocardium in renovascular hypertensive rats.
实验结果表明粉防己碱可部分逆转肾血管性高血压所致的大鼠左心室肥厚，降低心肌胶原含量，升高肌球蛋白ATP酶活性。
The results suggest that Tet or Ena partially reduce the hydroxyproline content and elevate myosin ATPase activity of hypertrophied myocardium in renovascular hypertensive rats.
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