目的探讨肾素血管紧张素醛固酮系统在甲亢性心脏病发病中的作用。
Objective To study the role of renin angiotensin aldosterone system in the pathogenesis of hyperthyroid heart disease.
结论肾素血管紧张素醛固酮系统激活是甲亢性心脏病发病的重要原因之一。
Conclusion Hyperthyroidism results in activation of renin angiotensin aldosterone system which contributes to the pathogenesis of hyperthyroid heart disease.
目的研究药物干预对高盐复合冷应激诱导大鼠的肾素血管紧张素醛固酮系统、血压和心肌肥厚的影响。
Objective To observe the effects of antihypertensive agents on renin angiotensin aldosterone system, blood pressure and myocardial hypertrophy induced by high salt diet plus cold stress.
目的探讨充血性心力衰竭患者血清钠水平对肾素血管紧张素醛固酮系统和心钠素(ANP)活性的影响。
Objective To investigate the effects of serum sodium on the activation of renin-angiotensin-aldosterone system and atrial natriuretic peptide (ANP) in patients with congestive heart failure.
结论异丙酚降压作用与其作用于KATP通道、抑制肾素活性，进而抑制肾素血管紧张素醛固酮系统有关。
Conclusion Propofol could decrease blood pressure, which may be due to its effect on the ATP sensitive KATP channels which could inhibit plasma renin activity (PRA) and RAAS.
结论充血性心衰患者宜适当补盐以维持正常血钠水平，这将有利于肾素血管紧张素醛固酮系统和ANP之间的平衡。
Conclusion an appropriate supplement of sodium can maintain the balance between the activity of renin-angiotensin-aldosterone system and ANP in patients with congestive heart failure.
背景：压力负荷可以导致肾素血管紧张素醛固酮系统激活，进而导致左室重构，卡托普利对这种重构具有逆转作用。
BACKGROUND: Pressure overload can lead to the activation of renin-angiotensin-aldosterone system, and then left ventricular remodeling occurs. Captopril can reverse this progress.
在肾素-血管紧张素-醛固酮系统中，肾小球旁体帮助调节血容量和压力。
In the renin - angiotensin - aldosterone system, the juxtaglomerular apparatus helps regulate volume and pressure.
肾素- 血管紧张素-醛固酮系统在血压昼夜节律的调节中起着关键作用。
The renin-angiotensin-aldosterone system plays an important role in regulating the circadian rhythms of blood pressure.
肾素-血管紧张素-醛固酮系统反应活跃，一般以偏高居多。
But the renin angiotensin aldosterone system had a very active, mainly hyperactivating, response.
开心胶囊可以抑制胶原蛋白的合成，增加心肌顺应性。 对肾素—血管紧张素—醛固酮系统的影响可能是其作用机制之一。
KC can inhibit the production of collagen and increase myocardial compliance and its mechanism may be involved in the regulation of rennin angiotensin aldosterone system.
结论糖尿病肾病及植物神经病变可累及肾-素血管紧张素-醛固酮系统，易造成低醛固酮血症。
Conclusion Diabetic nephropathy and autonomic neuropathy can affect renin, angiotensin and aldosterone system, and is easy to cause hypoaldosteronemia.
现综述近年来肾素-血管紧张素-醛固酮系统昼夜节律的机制及其对血压影响的研究进展。
This article reviews recent advances in research into the circadian rhythms mechanism of renin-angiotensin-aldosterone system and its influence on the circadian rhythm of blood pressure.
改善舒张功能可能与抑制肾素-血管紧张素-醛固酮系统(RAAS)过度激活、降低血液粘稠度及改善微循环等间接作用有关。
Improving diastole function may connected with restraining over-activation of RAAS, reducing blood density and improving micro-circulation and so on indirectly.
肾素血管紧张素 -醛固酮轴的活化导致增加。
The activation of the renin angiotensin–aldosterone axis leads to an increase.
肾素-血管紧张素-醛固酮系统（RAAS）是一种能被容量丢失激活的反馈控制系统。
The renin-angiotensin-aldosterone system (RAAS) is a feedback system activated with volume depletion.
高血压左心室肥厚(LVH)与肾素-血管紧张素-醛固酮系统及交感神经-儿茶酚胺系统的关系比较明确。
The relationship between hypertensive left ventricular hypertrophy (LVH) and renin-angiotensin-aldosterone system (RAAs) the sympathetic nervous-catecholamine (ca) system are clear.
高血压左心室肥厚(LVH)与肾素-血管紧张素-醛固酮系统及交感神经-儿茶酚胺系统的关系比较明确。
The relationship between hypertensive left ventricular hypertrophy (LVH) and renin-angiotensin-aldosterone system (RAAs) the sympathetic nervous-catecholamine (ca) system are clear.
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