肾活检3例均为急性肾小管坏死。
如果不进行治疗，肌红蛋白尿就可能造成肾小管坏死。
Myoglobinuria may result in renal tubular necrosis if untreated.
肾脏病理为急性肾小管坏死，肾间质炎症细胞浸润。
Renal biopsy revealed acute tubular necrosis and renal interstitial inflammation.
光镜下肾组织中白细胞（WBC）和肾小管坏死数目；
WBC in kidney hssue and necrotic tubules were counted under light microscope;
蛇毒直接导致肾灌注压改变、肾小球病变、肾小管坏死。
Venom directly results in the changes in renal perfusion pressure, glomerular lesion, and renal tubular necrosis.
方法急性肾小管坏死是肾移植术后较严重而常见的并发症。
Methods Acute tubular necrosis (ATN) is a common and Severe complication of renal transplantation.
结论术后影响移植肾成功的主要原因是急性排斥反应及急性肾小管坏死。
Conclusion The major causes of dysfunction of the transplanted kidney in the postoperative sfage are acute rejection and ATN.
急性缺血再灌注引起急性肾小管坏死发生机制较为复杂，相关因素比较多。
The mechanism of the acute tubular necrosis (ATN) caused by acute renal ischemia-reperfusion injury is complicated and there are many correlation factors in it.
目的：探讨尸肾移植术后急性肾小管坏死（ATN）的发病因素和防治措施。
Purpose:To discuss the pathogenic factors, treatment and diagnosis of acute tubular necrosis(ATN) following cadaveric kidney transplantation.
如果在围手术期给药钙通道阻断剂有减少移植后急性肾小管坏死(atn)的可能性。
Calcium channel blockers have the potential to reduce the incidence of post-transplant acute tubular necrosis (ATN) if given in the peri-operative period.
肾灌注严重减少则伤肾，导致急性肾小管坏死和肾机能不全，表现为少尿和血清肌酸酐的不断上升。
The kidneys are injured when renal perfusion is critically reduced, leading to acute tubular necrosis and renal insufficiency manifested by oliguria and progressive rise in serum creatinine.
肾灌注严重减少则伤肾，导致急性肾小管坏死和肾机能不全，表现为少尿和血清肌酸酐的不断上升。
The kidneys are injured when renal perfusion is critically reduced leading to acute tubular necrosis and renal insufficiency manifested by oliguria and progressive rise in serum creatinine.
目的探讨庆大霉素诱发的中毒性急性肾小管坏死动物模型的建立，研究其在基础及临床科研中的价值。
Objective to discuss the foundation of the animal model of gentamicin-induced toxic acute tubular necrosis and approach its value scientific research.
对比分析声学造影移植肾急性排斥和急性肾小管坏死血流灌注变化特点，探讨该方法对移植肾并发症鉴别诊断的可能性。
To analyse changes of blood perfusion ar and ATN comparison by contrast ultrasound and explore the feasibility of distinguishing diagnoses of renal allograft complication using the new method.
结果：移植肾术后早期少尿或无尿患者原因，急性排斥反应55.9%，急性肾小管坏死38.2%，加速排斥反应5.9%。
Results The reasons that led to oliguria or anuria early after transplantation were acute rejective reaction (55.9%), acute renal tubular necrosis (38.2%) and accelerated rejective reaction (5.9%).
肾动脉钳闭引起的肾脏缺血再灌注以及甘油、庆大霉素、氯化汞均可致实验动物急性肾衰竭，病理学上可见典型的急性肾小管坏死。
Animal ARF can be induced by renal ischemia-reperfusion injury, glycerine, gentamicin and mercuric chloride, and typical acute tubular necrosis can be observed in pathological changes.
而肾组织的病理改变主要表现为肾小管的坏死、脱落，管型形成，肾小球缺血、皱缩，肾组织内见多量pmn滞留。
The histopathological changes of the kidneys included renal tubular necrosis and detachment, formation of casts, glomerular ischemia and shrinkage and infiltration of large amount of PMN in LPS group.
肝细胞、肾小管细胞呈现溶解性坏死。
The liver cell and renal tubular cell showed to dissolve putrescence.
最右边是正常肾脏，其左边是濒临坏死的充血区，再往左边是苍白、粉红的梗死区，此处肾小球和肾小管都已坏死。
At the far right is normal kidney, then to the left of that hyperemic kidney that is dying, then to the left of that pale pink infarcted kidney in which both tubules and glomeruli are dead.
最右边是正常肾脏，其左边是濒临坏死的充血区，再往左边是苍白、粉红的梗死区，此处肾小球和肾小管都已坏死。
At the far right is normal kidney, then to the left of that hyperemic kidney that is dying, then to the left of that pale pink infarcted kidney in which both tubules and glomeruli are dead.
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