• 活检3例急性肾小管坏死

    Renal biopsy revealed acute tubular necrosis.

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  • 如果不进行治疗,肌红蛋白尿可能造成小管坏死

    Myoglobinuria may result in renal tubular necrosis if untreated.

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  • 病理急性肾小管坏死炎症细胞浸润。

    Renal biopsy revealed acute tubular necrosis and renal interstitial inflammation.

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  • 光镜组织中白细胞WBC肾小坏死数目

    WBC in kidney hssue and necrotic tubules were counted under light microscope;

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  • 蛇毒直接导致灌注改变肾小病变小管坏死

    Venom directly results in the changes in renal perfusion pressure, glomerular lesion, and renal tubular necrosis.

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  • 方法急性肾小管坏死移植术后严重常见并发症

    Methods Acute tubular necrosis (ATN) is a common and Severe complication of renal transplantation.

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  • 结论术后影响移植成功主要原因急性排斥反应急性小管坏死

    Conclusion The major causes of dysfunction of the transplanted kidney in the postoperative sfage are acute rejection and ATN.

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  • 急性缺血再灌注引起急性小管坏死发生机制较为复杂相关因素比较

    The mechanism of the acute tubular necrosis (ATN) caused by acute renal ischemia-reperfusion injury is complicated and there are many correlation factors in it.

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  • 目的探讨移植术后急性肾小管坏死ATN发病因素和防治措施。

    Purpose:To discuss the pathogenic factors, treatment and diagnosis of acute tubular necrosis(ATN) following cadaveric kidney transplantation.

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  • 如果手术期给药通道阻断减少移植后急性肾小管坏死(atn)可能性

    Calcium channel blockers have the potential to reduce the incidence of post-transplant acute tubular necrosis (ATN) if given in the peri-operative period.

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  • 灌注严重减少则导致急性肾小管坏死机能不全表现少尿血清肌酸酐的不断上升。

    The kidneys are injured when renal perfusion is critically reduced, leading to acute tubular necrosis and renal insufficiency manifested by oliguria and progressive rise in serum creatinine.

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  • 灌注严重减少则导致急性肾小管坏死机能不全表现少尿血清肌酸酐的不断上升。

    The kidneys are injured when renal perfusion is critically reduced leading to acute tubular necrosis and renal insufficiency manifested by oliguria and progressive rise in serum creatinine.

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  • 目的探讨庆大霉素诱发中毒性急性肾小管坏死动物模型建立,研究基础及临床科研中的价值

    Objective to discuss the foundation of the animal model of gentamicin-induced toxic acute tubular necrosis and approach its value scientific research.

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  • 对比分析声学造影移植急性排斥急性肾小管坏死血流灌注变化特点,探讨方法对移植并发症鉴别诊断可能性

    To analyse changes of blood perfusion ar and ATN comparison by contrast ultrasound and explore the feasibility of distinguishing diagnoses of renal allograft complication using the new method.

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  • 结果:移植术后早期尿尿患者原因急性排斥反应55.9%,急性小管坏死38.2%,加速排斥反应5.9%。

    Results The reasons that led to oliguria or anuria early after transplantation were acute rejective reaction (55.9%), acute renal tubular necrosis (38.2%) and accelerated rejective reaction (5.9%).

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  • 动脉钳闭引起缺血再灌注以及甘油庆大霉素氯化汞均致实验动物急性衰竭,病理学可见典型的急性肾小管坏死

    Animal ARF can be induced by renal ischemia-reperfusion injury, glycerine, gentamicin and mercuric chloride, and typical acute tubular necrosis can be observed in pathological changes.

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  • 组织病理改变主要表现为坏死脱落形成肾小缺血皱缩组织内见多pmn滞留。

    The histopathological changes of the kidneys included renal tubular necrosis and detachment, formation of casts, glomerular ischemia and shrinkage and infiltration of large amount of PMN in LPS group.

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  • 细胞小管细胞呈现溶解坏死

    The liver cell and renal tubular cell showed to dissolve putrescence.

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  • 右边正常左边濒临坏死充血区,往左边是苍白、粉红梗死区此处肾小坏死

    At the far right is normal kidney, then to the left of that hyperemic kidney that is dying, then to the left of that pale pink infarcted kidney in which both tubules and glomeruli are dead.

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  • 右边正常左边濒临坏死充血区,往左边是苍白、粉红梗死区此处肾小坏死

    At the far right is normal kidney, then to the left of that hyperemic kidney that is dying, then to the left of that pale pink infarcted kidney in which both tubules and glomeruli are dead.

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