疾病的最后的舞台当一个容器是侵蚀的由时，到达了疾病过程并且在肺以内的出血生产了濒死死亡。
The final stage of the disease arrived when a vessel was eroded by the disease process and hemorrhage within the lungs produced the agonal death.
目的研究肺表面活性蛋白A（SP-A）在肺出血新生儿支气管肺泡灌洗液（BALF）中的变化及其与预后的关系。
Objective To study the change of surfactant protein A(SP-A)in bronchoalveolar lavage fluid(BALF)and the relationship with prognosis in newborns with pulmonary hemorrhage.
这也是一个由于肺中动脉分支的血栓栓塞所引起的肺出血性梗死。
Here is another hemorrhagic pumonary infarction in a patient with a pulmonary thromboembolus to a medium sized pulmonary artery.
感染者有多种临床表现，其中重者表现为致死性肺弥漫性出血。
The infection causes a diversity of clinical manifestations, including fatal pulmonary diffuse hemorrhage.
大鼠的肺也出现充血、水肿、出血。
统计两组相关指标：平均穿刺时间、活检阳性率、肺内针道出血发生率及气胸发生率。
Positive rate of biopsy, mean puncture time, incidence of intrapulmonary hemorrhage and incidence of pneumothorax were also calculated.
肺梗死是出血性梗死，由于肺是双重血液供应的，支气管动脉可继续供血，但不能阻止梗死的发生。
A pulmonary infarct is hemorrhagic because of the dual blood supply from the non-occluded bronchial arteries which continue to supply blood, but do not prevent the infarction.
特发性肺含铁血黄素沉着症是一种原因不明的少见的肺出血性疾病。
Idiopathic pulmonary hemosiderosis is a rare pulmonary hemorrhagic disease dial pathogenetic cause is not clear.
组镜下见肺灶性出血，间质、肺泡水肿，大量炎细胞浸润。
Local hemorrhage, interstitial edema, alveoli exudation and inflammatory cells were observed in lungs in group a.
这也是一个由于肺中动脉分支的血栓栓塞所引起的肺出血性梗死。
Here is another hemorrhagic pulmonary infarction in a patient with a pulmonary thromboembolism to a medium sized pulmonary artery.
未发生全身出血；1例发生亚肺段性肺栓塞。
探讨高血压脑出血术后早期肺叩打对肺部感染的影响。
Objective: to probe into influence of early lung patting on pulmonary infection of postoperative hypertensive cerebral hemorrhage.
对大鼠肺组织含水量、肺血管通透性及肺出血量进行测定。
The lung water content(LWC), pulmonary vascular permeability (PVP) and hemorrhagic amount(PHA) were measured.
结果提示：并发于急性出血性胰腺炎的肺损伤以肺泡表面活性物质系统受损为一重要的表现形式。
The results suggest that the lung injury resulting from acute hemorrhagic pancreatitis was characterized by alveolar surfactant System damage.
肺出血组肺毛细血管基质弹力纤维多处断裂，对照组阴性。
The elastic fibers of pulmonary capillary matrix broke in many place of group A but not in group B.
两组的平均穿刺时间、活检阳性率、肺内针道出血及气胸发生率无显著性差异。
Mean puncture time, positive rate of biopsy, incidence of intrapulmonary hemorrhage and incidence of pneumothorax had no significant difference between two groups.
病变中肺泡水肿、肺出血及肺充血以HTP组最严重，肺组织坏死及炎性细胞浸润以HTNP及HTP组最明显。
The alveolar edema and lung hemorrhage were the severest in the HTP group. The lung necrosis and the inflammatory cells infiltration were the severest in the HTP and HTNP group.
结果院内死亡1例（0.76%），主要并发症：出血，低心排出量综合征，心包填塞，心律失常，肺炎肺不张。
Results- The hospital mortality was (0.76%) (1/131), The leading complications were bleeding, low cardiac output syndrome, pericardial tamponade, cardiac arrhythmia , pneumonia and atelectasis.
死亡动物肺充血、出血甚至肺部呈红肝样变；
There were hyperemia, hemorrhage in lungs and pulmonary consolidation;
结果被检病死小尾寒羊无特征性临床表现，剖检可见肺肿胀，肠黏膜脱落、出血等。
The characteristic symptoms were not observed, pathological examinations of diseased sheep exhibited swelling of lung, fall off and haemorrhages of intestinal mucous membrane.
结果撞击侧肺(右肺)发生明显的出血、充血、水肿，光镜下可见肺组织较广泛的出血和结构破坏。
Results the right lung showed obvious hyperaemia, congestion and edema and with extensive blood extravasation and destruction of tissues, but only edema was seen in the left lung.
目的探讨LPS致新生大鼠肺损伤以至于肺出血的发病机制。
Objective To investigate the pathophysiology of acute lung injury in newborn rats induced by lipopolysaccharide.
目的探讨LPS致新生大鼠肺损伤以至于肺出血的发病机制。
Objective To investigate the pathophysiology of acute lung injury in newborn rats induced by lipopolysaccharide.
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