MASSON染色观察肺组织纤维化程度；
MASSON dyeing observation lung organization fibrosis degree;
结果表明：国产蓝棉能引起大鼠肺组织纤维化。
The results showed that Chinese crocidolite asbestos was able to induce lung fibrosis in rats.
特发性肺纤维化以进行性肺损伤、炎症、肺组织纤维化为特征。
IPF is characterized by progressive injury, inflammation, and fibrosis of the lung parenchyma.
游离二氧化硅致肺组织纤维化机制十分复杂，涉及多种细胞及生物活性物质参与的炎症反应、免疫反应、异物反应、细胞毒作用等。
The mechanism of fibrosis caused by free silica particles involves a lot of cells and reactions including inflammatory reaction, immunological reaction, foreign body reaction and cell toxic action.
这两名孕妇均死于由肺组织严重纤维化所引发的多器官衰竭。
The expectant women died of multiple organ failure triggered by severe scarring and thickening of the lung tissue.
与之相应的肺组织病理改变为早期的急性肺泡炎和后期纤维化改变。
The above changes accompanied with lung pathological manifestations, which included acute alveolitis during the earlier period and lung fibrosis during later period.
将这一方法与肺组织羟脯氨酸含量测定相比较，表明二者在显示肺纤维化程度和变化趋势上具有相同的结果。
Compared with lung hydroxyproline content measurement, it was shown that both have a similar result in displaying the extent of pulmonary fibrosis and its alteration trend.
目的检测结缔组织生长因子(CTGF)在大鼠移植肺内的表达，并探讨其与移植肺纤维化关系。
Objective to investigate the role of connective tissue growth factor (CTGF) in lung allograft rejection in rats.
方法采用大鼠油酸尾静脉注射制作急性肺损伤后肺纤维化模型，观察红霉素对损伤肺组织中中性粒细胞聚集及氧自由基的干预作用。
Methods: Model of fibrosis of lung with injection by oil-acid to rat, observed the effect of erythromycin to neutrophil and oxygen-free radical.
肺纤维化是指多种原因导致的，以弥漫性肺间质组织增生为特征的疾病。
Pulmonary fibrosis is caused by many factors and shows diffuse interstitial tissue hyperplasia.
结果：实验组经博莱霉素诱导4周后，取肺组织行HE染色，病理符合肺纤维化改变；
Results After 4 weeks induction by bleomycin solutions, the lung tissue was obtained and stained by HE, the pathological changes were in accordance with pulmonary fibrosis.
结论三棱和莪术能减少肺组织细胞过度凋亡，从而延缓肺纤维化进程。
Conclusion Rhizoma Sparganii and Rhizoma Curcumae can delay the fibrosis of lung tissue through reducing excessive apoptosis of lung tissue.
方法：羟脯氨酸法、间接酶联免疫吸附法（ELISA）测定博莱霉素A6（BLMA6）诱发的小鼠、大鼠肺纤维化模型肺组织胶原含量及合成，阿利新蓝法测定其糖胺多糖（GAG）含量；
METHODS: Hydroproline determination and ELISA were used for measuring the content of collagen, Alcian blue assay for BLM-A6 induced glycosaminoglycan (GAG) in the lung tissues (rats, mice).
结论：当归多糖对肺纤维化大鼠的肺功能、肺组织病理学改变、细胞外基质代谢等均具有明显的改善作用。
Conclusion: Angelica polysaccharide has made great improvement in the changes of pulmonary function, pathological morphology, extracellular stroma metabolism and etc. in lung fiberized rats.
尽管已知吸烟会引起肺内免疫细胞的募集和组织纤维化，然而吸烟如何导致这些变化却不清楚。
Although lung exposure to cigarette smoke leads to immune cell recruitment and tissue fibrosis, how cigarette smoke causes these changes is largely unknown.
结果肺组织病理形态学观察发现模型组小鼠肺泡炎及纤维化程度明显高于阴性对照组；
Results The degree of airsacculitis and fibrosis in model group are obviously higher than the negative control group by pathological observation.
将这一方法与肺组织羟脯氨酸含量测定相比较，表明二者在显示肺纤维化程度和变化趋势上具有相同的结果。
Compared with lung hydroxyproline content measurement, it was shown that both have a similar result in displaying the extent of pulmonary fib...
通过对肺组织HE染色及电脑图像分析观察肺纤维化的程度；
We dyed the pulmonary fibrosis tissue with HE and utilized computer to reflect the degree of PF objectively by image pattern analysis.
通过对肺组织HE染色及电脑图像分析观察肺纤维化的程度；
We dyed the pulmonary fibrosis tissue with HE and utilized computer to reflect the degree of PF objectively by image pattern analysis.
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