目的探讨放射性肺损伤的影像学表现以及病理发生机制。
Objective To investigate the imaging features of radiation-induced pulmonary injury and the pathology mechanism.
目的探讨胆红素对内毒素致急性肺损伤(ALI)的保护作用及其可能机制。
ObjectiveTo investigate the effects of of bilirubin on LPS-induced acute lung injury (ALI) in rats and its possible mechanisms.
目的：探讨山莨菪碱（6542）对油酸引起的家兔急性肺损伤的防治作用及其机制。
Objective:To investigate the therapeutic and preventive effects of anisodamine(6542)on acute pulmonary injury induced by oleic acid in rabbits and their mechanism.
目的探讨腺苷预处理在心内直视手术中对肺缺血再灌注损伤的保护效果及其机制。
Objective To explore the protective effects and mechanism of adenosine preconditioning on pulmonary ischemia-reperfusion injury in patients undergoing open heart surgery.
目的：探讨兔肺移植中前列腺素a1 (PGA1)在对供肺缺血再灌注损伤的保护作用及其可能的作用机制。
Objective: To investigate the protective effects of prostaglandin A1 (PGA1) on ischemia-reperfusion during rabbit lung-transplant, and explore its possible mechanism.
本文就中性粒细胞弹力蛋白酶在肺组织损伤修复中的作用和机制进行综述。
In this review, the actions and mechanism of neutrophil elastase in the repairing lung injury were summarized.
急性肺损伤和急性呼吸窘迫综合征发病机制复杂，病死率高，是临床常见的呼吸系统急危重症。
Acute lung injury and acute respiratory distress syndrome are severely acute respiratory diseases and are common in clinic, and their pathogenesis is complicated and the mortality is high.
本实验中单肺原位移植造成健侧肺组织一定程度的损伤，但其原因和机制还有待于进一步的研究。
To a certain extent, single lung orthotopic transplantation has adverse effects on the contralateral lung. Its reason and mechanism is not clear and further investigation is expected.
本实验研究了654-2对油酸所致离体血管内皮细胞直接损伤作用的影响，以探讨654-2改善油酸所致急性肺损伤的机制。
The effect of anisodamin (654-2) on oleic acid-induced injury of cultured bovine endothelial cells was studied to elucidate the mechanism of protective roles of anisodamin on acute lung injury.
结论：肠淋巴管结扎干预休克肺损伤的机制与上调或下调上述相关基因的表达有关。
CONCLUSION: the results indicate that the mechanism of mesenteric lymph ligation alleviating the lung injury after shock is related to up or down regulation of the above genes.
目的建立气管注入细菌致兔急性肺损伤(ALI)模型，为研究细菌致ali的致病机制及其治疗提供理论依据。
Objective: to establish the rabbit model of acute lung injury (ALI) by intratracheal instillation of Escherichia coli, and to study the pathogenesis of bacteria-induced ALI.
目的观察川芎嗪对肺缺血-再灌注损伤脂质过氧化的保护作用，探讨肺缺血-再灌注损伤的发病机制及川芎嗪的保护作用。
Objective to observe the protective effects of Ligustrazine on pulmonary ischemia-reperfusion in rabbits and to investigate the possible mechanisms of pulmonary ischemia-reperfusion injuries.
目的探讨内毒素肺损伤时，肺泡巨噬细胞逐步由免疫防御型转变为效应型的分子机制。
Objective To study the molecular mechanism of the conversion of alveolar macrophages (AMs) from immune defensive cells to effective cells in mice with endotoxin induced lung injury.
目的：探讨抗氧化剂治疗高氧肺损伤的疗效及机制。
Objective: To explore the mechanism and therapeutical effect of anti-oxidation on hyperoxic lung injury.
目的探讨肠缺血再灌流引起肺损伤的细胞机制。
Objective To investigate the relationship bet we en lung injury from intestinal ischemia-reperfusion and polymorphonuclear neutrophils in rats.
目的：采用大鼠肠缺血再灌注模型，对创伤休克后多系统器官衰竭中肺损伤的发生机制进行研讨。
Objecctive: To explore the pathogenesis of lung injury during multiple system organ failure (MSOF) after traumatic shock by using intestinal ischemia reperfusion model of the rat.
急性肺损伤(ALI)是体外循环(CPB)后的重要并发症之一，具体机制尚未完全清楚。
Acute lung injury (ALI) is one of the most important complications after cardiopulmonary bypass (CPB) and the complex pathophysiology remains to be resolved incomplete.
目的探讨痰热清注射液抗内毒素所致急性肺损伤的作用机制。
OBJECTIVE To study the effects of Tanreqing parenteral solution on acute lung injury induced by lipopolysaccharide.
结论：SND有抗大鼠肺缺血-再灌注损伤作用，其机制可能与清除氧自由基和改善组织灌流有关。
CONCLUSION: These results indicate that SND may have a protective effect on ischemia-reperfusion injured lung by its antioxidant activity and by adjusting NO level.
目的研究丙泊酚对内毒素(LPS)诱导的大鼠急性肺损伤(ALI)的保护作用及其可能分子作用机制。
Objective To study the protective effect of propofol on acute lung injury (ALI) induced by lipopolysaccharide (LPS) in rats.
目的探讨LPS致新生大鼠肺损伤以至于肺出血的发病机制。
Objective To investigate the pathophysiology of acute lung injury in newborn rats induced by lipopolysaccharide.
目的探讨胆红素对实验性大鼠肺缺血再灌注损伤的保护作用并探讨其发生的机制。
Purpose To study the mechanism of lung ischemia - reperfusion injury and the protective effects of Bilirubin on lung ischemia - reperfusion injury.
结论PD 142893能减轻大鼠肺移植早期缺血再灌注损伤，机制与减轻肺间质水肿及炎症反应有关。
Conclusion Treatment of lung allografts with PD142893 can ameliorate ischemia - reperfusion injury by alleviating lung interstitial edema and inhibiting inflammatory reaction.
急性肺损伤的病理机制复杂，临床和实验研究发现多种中药对其具有改善作用。
The pathogenesis of acute lung injury is complex. Increasing evidence indicates that many traditional Chinese medicines are beneficial in treatment of acute lung injury.
急性肺损伤的病理机制复杂，临床和实验研究发现多种中药对其具有改善作用。
The pathogenesis of acute lung injury is complex. Increasing evidence indicates that many traditional Chinese medicines are beneficial in treatment of acute lung injury.
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