作者采用山羊慢性肺淋巴瘘方法，观察缺氧、空气栓塞单独和复合作用，对清醒山羊肺动脉压、肺微血管壁通透性和肺内液体交换的影响。
The effects of hypoxia and air microembolism on pulmonary pressor response and lung fluid exchange were studied in eight conscious goats with chronic lung lymph fistula.
目的研究吸入一氧化碳(CO)对急性缺氧性肺动脉高压及缺氧性肺损伤的影响。
Objective To investigate the effect of carbon monoxide (CO) inhalation on acute hypoxic pulmonary arterial hypertension and acute hypoxic lung injury.
目的探讨一氧化碳对缺氧性肺血管结构重建大鼠肺动脉中转移生长因子(TGF)表达的影响。
Objective to investigate the impact of transforming growth factor (TGF) in the mechanisms by which carbon monoxide (CO) regulates hypoxic pulmonary vascular remodeling.
目的：观察肺心汤对慢性缺氧性肺动脉高压大鼠的作用。
Objective: To observe effects of Feixin Tang (FXT) on the chronic hypoxia pulmonary hypertension rats.
布洛芬抑制前列腺素的生成，并通过增强缺氧性肺血管收缩反应而改善了肺损伤时的气体交换。
The inhibition of prostaglandin production with Ibuprofen strengthened hypoxic pulmonary vasoconstriction thus improved pulmonary gas exchanges in acid lung injury.
结果提示：高原移居平原者与世居平原者相比，脑血管缺氧性扩张反应较强，而肺血管缺氧性收缩反应较弱。
This study suggests that long term residing at high altitude results in an increase in cerebral vasodilative response and a decrease in pulmonary vasoconstrictive response to acute hypoxia.
结论一氧化碳对缺氧性肺血管结构重建大鼠肺动脉中TGF表达有抑制作用。
Conclusion Carbon monoxide inhibited TGF expression by pulmonary arteries of rats with hypoxic pulmonary vascular remodeling.
结论一氧化氮对缺氧性肺血管结构重建大鼠肺动脉中PDGF表达有抑制作用。
Conclusion Nitric oxide inhibited PDGF expression by pulmonary arteries of rats with hypoxic pulmonary vascular remodeling.
目的探讨慢性缺氧引起缺氧性肺血管收缩反应降低的机制。
Objective To investigate the mechanism of chronic hypoxia induced decreased HPV.
作者研究了慢性间断低压缺氧对大鼠肺血管反应性的影响及其与组胺的关系。动物经慢性缺氧后发生慢性肺动脉高压，其肺血管对急性缺氧的反应性降低。
The effect, of chronic intermittent hypobaric hypoxia on pulmonary vascular reactivity to acute hypoxia in. rats and its relationship to histamine have been studied.
缺氧时肺动脉压显著升高，这可能是肺毛细血管前肌性小动脉收缩的结果。
Hypoxia produced a sustained increase in pulmonary arterial pressure (Ppa), the site of vasoconstriction was proximal to the lung capillaries.
缺氧时肺动脉压显著升高，这可能是肺毛细血管前肌性小动脉收缩的结果。
Hypoxia produced a sustained increase in pulmonary arterial pressure (Ppa), the site of vasoconstriction was proximal to the lung capillaries.
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