利用细胞化学方法，通过图象分析仪，对吞噬煤尘后的肺巨噬细胞内酸性磷酸酶活性进行了定量观察。
The activity of acid phosphatase (ACP) in lung macrophages of rats with coal dust ingestion was observed by cell chemical method and by image analyser.
从超微结构方面研究了动物模型中三个煤种的煤尘与肺巨噬细胞的相互作用及其作用下的肺组织反应。
The ultrastructural changes of lungs tissue reaction of three kinds of dusts and their interaction with alveolar macrophages were studied in animal models.
结论亚急性BHT大剂量染毒可造成大鼠肺、肝等脏器功能性和器质性损伤，BHT可诱发肺巨噬细胞凋亡的产生。
Conclusion BHT may cause functional and organic damage of lung and liver, and it can induce apoptosis to pulmonary macrophage in rats.
结论肺内淋巴液的生成可能以小泡转运系统为主，肺巨噬细胞及白细胞迁移入淋巴管的通道可能是位于细胞连接处的一侵蚀性通道。
Conclusion Vesicles transportation might be the main pathway of lymph formation in lung, the lymphatic passage through which pulmonary macrophage and leukocyte pass might be a erosive passage.
结果实验组肺内炎症细胞增多，以巨噬细胞和淋巴细胞为主。
Results In experiment group, there were increased inflammatory cells in lung, and most of them were macrophage and lymphocyte.
结论禽流感病毒感染的食蟹猴肺组织内抗原提呈细胞主要以巨噬细胞为主。
Conclusion Macrophages are the main antigen presenting cells in the lung tissue of cynomolgus macaque infected with avian influenza virus.
巨噬细胞吞噬异物，如肺内的尘埃及碳粒，同样还有活体染料的微粒。
Macrophages phagocytize foreign materials such as dust and carbon particles in the lung, as well as vital dye particles.
目的探讨肺泡巨噬细胞活化在急性坏死性胰腺炎(ANP)肺损伤中的作用。
Objective To discuss the role of alveolar macrophage activation in rats with acute necrotizing pancreatitis (ANP) associated with lung injury.
目的探讨肺腺癌肿瘤相关巨噬细胞(TAM)浸润与淋巴管生成、患者生存的关系。
Objective To examine the relationship between tumor-associated macrophages (TAMs) infiltration and the density of lymphangiogenesis or the patients' clinical outcome in lung adenocarcinoma.
目的探讨内毒素肺损伤时，肺泡巨噬细胞逐步由免疫防御型转变为效应型的分子机制。
Objective To study the molecular mechanism of the conversion of alveolar macrophages (AMs) from immune defensive cells to effective cells in mice with endotoxin induced lung injury.
结核菌存在于肺中，在免疫细胞中称巨噬细胞。
M. tuberculosis lives in the lungs, in immune cells called macrophages.
目的：初步探讨八肽胆囊收缩素(CCK - 8)对体外脂多糖(LPS)激活大鼠肺间质巨噬细胞(IM)的调节作用。
AIM: to study the effect of cholecystokinin octapeptide (CCK-8) on lipopolysaccharide (LPS) -stimulated pulmonary interstitial macrophage (im) in vitro.
采用体外细胞实验方法，观察了石英尘对DPH和ANS标记的肺胞巨噬细胞膜流动性和荧光性质的影响，同时测定了肺泡巨噬细胞活性氧的产生。
The effects of quartz on membranous fluidity labelled with DPH and ANS and the production of oxygen free radicals of alveolar macrophages were studied in vitro.
目的：探讨慢性阻塞性肺疾病(COPD)模型大鼠肺泡巨噬细胞延迟整流钾通道(KV)活性变化。
AIM: To explore the change of delayed rectifier potassium channel (KV) activity in alveolar macrophages (AM) in chronic obstructive pulmonary disease (COPD) rats.
目的：探讨慢性阻塞性肺疾病(COPD)模型大鼠肺泡巨噬细胞延迟整流钾通道(KV)活性变化。
AIM: To explore the change of delayed rectifier potassium channel (KV) activity in alveolar macrophages (AM) in chronic obstructive pulmonary disease (COPD) rats.
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