目的：研究三羟异黄酮对肠缺血再灌注损伤的影响。
Objective: To investigate the effects of genistein on intestinal ischemia-reperfusion injury in rats.
目的：探讨川芎嗪对家兔肠缺血再灌注损伤作用的机理。
Objective: To research the effect of tetramethylpyrazine (TMP) on rabbit intestinal ischemic reperfusion.
结 果：肠缺血再灌注损伤后，肝、肾功能的生化指标升高；
Results: After intestinal ischemia reperfusion injury, the biochemistry increased which indicated that function of liver and kidney was damaged.
本文概述了肠缺血再灌注损伤的可能机理及其预防和治疗措施。
This review was performed to summarize the possible mechanism and therapy in ischemia reperfusion injury of intestine.
结论：诱导HO - 1可通过抑制细胞凋亡而减轻移植肠缺血再灌注损伤。
Conclusion: HO-1 can alleviate the ischemia-reperfusion injury of transplanted small bowel by reducing apoptosis.
目的：探讨超氧化物歧化酶(SOD)及川芎嗪(CXZ)对肠缺血再灌注损伤的作用。
Objective To research the effect of SOD and CXZ on rabbit intestinal ischemic reperfusion.
背景：肠道因素尤其是肠缺血再灌注可导致远隔器官损伤是创伤。
BACKGROUND: intestinal tract factors, especially intestinal ischemia-reperfusion, can induce the injury of remote organ.
目的探讨人工生物膜对大鼠肠缺血再灌注所致小肠及肝脏损伤的保护作用。
Objective To explore the protective effects of artificial membrane on functional injury of intestine and liver after gut ischemia-reperfusion in rats.
目的观察丙泊酚对大鼠肠缺血再灌注（I/R）后肠组织损伤的影响。
Objective To investigate the effect of propofol on gut injury following intestinal ischemia-reperfusion(I/R).
目的：探讨肠缺血再灌注（IR）损伤对远隔器官肺脏的损伤机制。
Objective:To investigate the injury mechanism concerning acute lung injury associated with local intestinal ischemiareperfusion(IR).
目的：采用大鼠肠缺血再灌注模型，对创伤休克后多系统器官衰竭中肺损伤的发生机制进行研讨。
Objecctive: To explore the pathogenesis of lung injury during multiple system organ failure (MSOF) after traumatic shock by using intestinal ischemia reperfusion model of the rat.
目的：观察灯盏花注射液对小鼠肠缺血再灌注肝损伤生化指标的影响。
Objective: to observe the effects of erigeron injection on the biochemical indicators of hepatotoxicity caused by ischemia and reperfusion of the mice intestine.
目的：观察灯盏花注射液对小鼠肠缺血再灌注肝损伤生化指标的影响。
Objective: to observe the effects of erigeron injection on the biochemical indicators of hepatotoxicity caused by ischemia and reperfusion of the mice intestine.
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