结论：大鼠饥饿后早期确有肠黏膜组织结构受损，发生内毒素移位，同时伴有肠黏膜免疫学屏障受损。
CONCLUSION: the structure of intestinal mucosa is damaged in the earlier stage of starvation in rats, accompanied by endotoxin translocation and dysfunction of intestinal mucosal immune barrier.
该文应用PAF受体拮抗剂研究PAF对幼年大鼠肠黏膜免疫屏障功能损伤作用。
This study aimed to investigate the effect of PAF on the injury of intestinal mucosal immuno-barrier function in young rats.
肠道菌群具有促进肠黏膜免疫系统生长与发育和调控肠黏膜屏障与免疫功能的双重作用。
The intestinal flora plays an important role both in promoting growth and development of the intestinal mucosal immune system and in regulating intestinal mucosal barrier and immune functions.
肠道菌群具有促进肠黏膜免疫系统生长与发育和调控肠黏膜屏障与免疫功能的双重作用。
The intestinal flora plays an important role both in promoting growth and development of the intestinal mucosal immune system and in regulating intestinal mucosal barrier and immune functions.
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