肥胖抵抗大鼠与肥胖大鼠都有肝脏脂肪变性与胰腺组织受损；
There were fatty degeneration and pancreatic tissue damage in both DIO-R rats and DIO rats.
作者认为NAFLD是超声扫描下肝脏脂肪变性最常见的致病因素（81.5%）。
The authors note that NAFLD was the most common cause (81.5%) of hepatic steatosis on ultrasound.
肝脏胰岛素抵抗还表现为脂代谢异常，如甘油三酯释放增加和肝脏脂肪变性，这与FOXO1转录活性增加有密切关系。
Increased triglyceride release and hepatic steatosis are the common features of hepatic insulin resistance, which deeply associated with increased FOXO1 transcription.
结果发现，肝脏脂肪变性与急性严重感染有密切关系，其脂肪变性主要与感染、毒素等损害了肝细胞内线粒体及其酶的活性有关。
The results showed that liver steatosis was closely related to the serious acute infection, in which the infection or toxin might damage enzyme and thread granules in the liver cells.
纳米态红色元素硒对小鼠的肝脏充血、浊肿、脂肪变性、坏死病理变化有明显改善作用。
Nano red elemental selenium obviously alleviated the histopathologic changes including liver hyperaemia, Claude swelling, fatty degeneration and necrosis.
纳米态红色元素硒对小鼠的肝脏充血、浊肿、脂肪变性、坏死病理变化有明显改善作用。
Nano red elemental selenium obviously alleviated the histopathologic changes including liver hyperaemia, Claude swelling, fatty degeneration and necrosis.
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