目的观察肝脂消合剂对小鼠肝脂肪变性的治疗作用。
Objective To observe treatment effects of Ganzhixiao Mixture on the fatty denaturation of mice liver.
目的探讨熊胆粉对高脂饮食豚鼠肝脂肪变性的预防及其作用机理。
Objective To determine whether bear bile could prevent fatty degeneration of liver cells caused by high fat diet and the mechanism.
脂肪性肝病是多种原因引起的病变主体在肝小叶、以肝细胞脂肪变性和脂肪蓄积过多为主的临床病理综合征。
Fatty liver disease (FLD) is a clinicopathological syndrome caused by a variety of reasons. The main lesions are in hepatic lobule with steatohepatitis and accumulation of triglycerides in the liver.
了解hCV核心蛋白在诱发肝脂肪变性中的分子学机制，对丙型肝炎相关性脂肪肝的病因学，预防和治疗等方面具有极大的促进作用。
Understanding the molecule mechanisms of HCV core protein induction hepatic steatosis may contribute to elucidate the etiology, prevention and treatment of HCV hepatic diseases.
方法：取正常人肝l - 02细胞，和经50%胎牛血清造模24小时建立的脂肪变性肝细胞模型，共同作为研究对象。
Methods: Use L-02 hepatocyte of normal human and the hepatocyte under fatty change after exposure to 50% fetal bovine serum for 24 hours as study objects.
结论：NHBT延长是引起IPGF的主要危险因素，供肝脂肪变性、CIT、RWIT延长可能是潜在危险因素。
Conclusion: NHBT is an important risk factor leading to IPGF while steatosis in donor liver. CIT and RWIT are potential risk factors.
结论：NHBT延长是引起IPGF的主要危险因素，供肝脂肪变性、CIT、RWIT延长可能是潜在危险因素。
Conclusion: NHBT is an important risk factor leading to IPGF while steatosis in donor liver. CIT and RWIT are potential risk factors.
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