B71的表达与肝组织炎症活动程度及纤维化进程密切相关。
B7 1 expression was closely correlated with the histological activity grade and the progress of hepatic fibrosis.
四项指标与肝组织炎症坏死及纤维化程度均呈显著相关（P<0.05）。
There were significant correlation(P<0.05) between the four indexes and the degree of inflammation, necrosis and fibrosis.
结论运用血清学肝纤维化指标评价肝组织纤维化程度时应考虑肝组织炎症程度对肝纤维化指标的影响。
Conclusion the influence of liver inflammation to serum fibrosis markers must be considered when the liver fibrosis staging is evaluated by these markers.
结果：慢性肝炎肝组织炎症活动程度越重或肝纤维化程度越明显则血清HA水平越高（P＜ 0．05或0．01）；
Results:There was a close correlation between serum HA levels and the grade of necroinflammatory activity and stage of fibrosis of CHB( P <0.05 or 0.01).
模型组大鼠肝小叶结构紊乱，有大量纤维组织沉积，炎症细胞浸润，肝细胞脂肪变性。
Lobules of liver in the rats of model group was disorder with a pile of deposition of fibrous tissue inflammatory cell infiltration and fatty degeneration of hepatocytes.
肝、肺组织内CD 14和SR表达变化与局部炎症反应失衡及器官损害有一定内在联系。
The expression of SR and CD14 in the liver and the lung showed intrinsic correlation with local inflammatory disequilibrium and organ injury.
结论：拉米夫定治疗慢性乙型肝炎，可以明显降低血清hbv - DNA及转氨酶水平，延缓肝组织纤维化的进展，减轻肝组织的炎症坏死。
Conclusion: Lamivudine treatment could decrease the HBV-DNA and transaminase level, and delay the progression of liver fibrosis and lighten inflammation and necrosis of liver.
肝组织学观察：模型对照组大鼠肝细胞炎症减轻，以致密较细的不完全纤维间隔为主。
Histopathology with lightened inflammation of hepatocytes and finer complete fibrous septums in model rats.
应用病理学判断肝组织的炎症及纤维化程度。
Liver inflammation and fibrosis was determined by Pathology.
同时肝组织经he染色，光镜下评估脂肪变性、炎症和坏死程度。
Meanwhile, the fatty degeneration, the degrees of inflammation and necrosis of liver samples were evaluated under the light microscope after he staining.
同时肝组织经he染色，光镜下评估脂肪变性、炎症和坏死程度。
Meanwhile, the fatty degeneration, the degrees of inflammation and necrosis of liver samples were evaluated under the light microscope after he staining.
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