软组织损伤通过检测血清肌酸磷酸激酶评价；
The soft-tissue injury was assessed by measuring serum creatine kinase.
同时测定肌酸磷酸激酶-MB型(CK - MB)的活性。
Serum creatine phosphokinase-MB (CK-MB) activity was detected.
脑出血后心电图异常，血清肌酸磷酸激酶B型(CKMB)改变及心脏损害均以血肿高峰期最为显著。
ECG abnormality, serum CK MB change and heart damage became significant during the peak time of intracerebral hematoma formation.
结果显示：控制性再灌注可减轻心肌超微结构损伤，降低心肌肌酸磷酸激酶释放及促进心脏功能的恢复。
Results suggested that control reperfusion could attenuate the damage of myocardial ultrastructure, decrease the release of CPK and improve cardiac functions.
血清肌酸磷酸激酶（CPK）活性均值病区组非常明显地高于改水病区组（P<0.01）和非病区组（P<0.01）。
In the non-defluoride group the mean of activity of urine creatine phosphokinase(CPK) was much higher than that in defluoride group ( P< 0.01) and non - epidemic area group (P<0.01) .
测定血液中肌酸磷酸激酶(CK)、乳酸脱氢酶(LDH)及丙二醛(MDA)和超氧化物歧化酶(SOD)含量以及心肌梗死范围。
The activity of creatine phosphokinase (CK), lactate dehydrogenase (LDH), and superoxide dismutase (SOD), the content of malondialdehyde (MDA) and the infarct size were investigated too.
测定血液中肌酸磷酸激酶(CK)、乳酸脱氢酶(LDH)及丙二醛(MDA)和超氧化物歧化酶(SOD)含量以及心肌梗死范围。
The activity of creatine phosphokinase (CK), lactate dehydrogenase (LDH), and superoxide dismutase (SOD), the content of malondialdehyde (MDA) and the infarct size were investigated too.
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