小鼠肌内埋置后植入部位无肌肉坏死，炎症反应轻，无纤维包裹。
The intramuscular implantation in mice induced no muscular necrosis or fibrous capsule formation, except for slight inflammatory reaction.
不可作肌肉注射以免引起肌肉坏死。静脉或皮下注射能引起溶血性贫血?血尿，蛋白尿。
Intra - muscular injection can lead to necrosis. intravenous or subcutaneous injection can cause hemolytic anemia, hematuria and proteinuria.
动态压缩几乎只发生在(97.5%)在冠状动脉左前降支完全包埋的病人，不论存不存在肌肉坏死。
Dynamic compression occurred almost exclusively (97.5% of the time) in patients with full encasement of the left anterior descending coronary artery, regardless of the presence of overlying muscle.
同时，拔定膏对无名的体外红肿痛肿块有较快的治疗过程，对于外伤引起骨头周边肌肉坏死有去毒修复作用。
At the same time, set out to unknown in vitro cream rapidly swelling pain mass course of treatment for traumatic cause bones surrounding muscle necrosis have poison to repair function.
此外，来自坏死肌肉的毒素突然释放进入循环系统，会造成肌红蛋白尿，如果不予治疗就会引起肾功能衰竭。
Further, the sudden release of toxins from necrotic muscle into the circulatory system leads to myoglobinuria, which causes renal failure if untreated.
普通心脏病发作时，患者动脉受阻，心脏肌肉得到不到含氧血，迅速导致组织坏死，并可能永久损害心脏功能。
In a conventional heart attack, an obstructed artery starves the heart muscle of oxygenated blood, quickly resulting in the death of tissue and potentially permanently compromising heart function.
目的：根据骨折伴软组织损伤患者的肌酸激酶测定值，判别肌肉损伤及坏死的程度。
Objective:To evaluate the degree of the injuries and necrosis of muscle based on serum creatine kinase values.
肌肉活检可见灶性肌纤维坏死，肌纤维横纹消失，小血管壁增厚，个别毛细血管内血栓形成。
Muscular biopsy revealed focal necrotic muscle with loss of muscle fibers, thickened walls of small blood vessels and occasionally thrombosis in capillary.
结论：小腿坏死肌肉彻底切除是动脉下段损伤延迟诊断后采取保肢治疗的重要措施；
Conclusion: It's an important step to resect the necrotic muscles in the limb salvage surgery for the popliteal artery inferior segment trauma with delayed diagnosis.
埋植材料周围肌肉组织无脓肿形成和组织坏死发生，局部组织反应为非细菌性炎症反应。
The biodegradation pattern in vivo was similar to that in vitro. There was no abscess formation and tissue necrosis surrounding the implanted materials.
目的：探讨肿瘤坏死因子相关诱导凋亡配体(trail)蛋白和顺铂协同抑制横纹肌肉瘤细胞生长和诱导凋亡作用及其机制。
AIM: to study the synergistic induction of apoptosis by TRAIL and cisplatin in rhabdomyosarcoma cells and investigate the role of mitochondrial membrane potential and caspase-3 in this process.
目的：探讨肿瘤坏死因子相关诱导凋亡配体(trail)蛋白和顺铂协同抑制横纹肌肉瘤细胞生长和诱导凋亡作用及其机制。
AIM: to study the synergistic induction of apoptosis by TRAIL and cisplatin in rhabdomyosarcoma cells and investigate the role of mitochondrial membrane potential and caspase-3 in this process.
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