目的：观察严重烧伤延迟复苏时大鼠心肌细胞线粒体呼吸功能的变化。
Objective: to investigate the changes of mitochondria respiratory function of myocardial cell in burnt rats after delayed fluid resuscitation.
结论：严重烧伤大鼠延迟复苏时会加重心肌细胞线粒体呼吸功能损伤。
Conclusion: Delayed fluid resuscitation results in more severe injury of mitochondria respiratory function of myocardial cell in burnt rats.
目的研究糖尿病大鼠小肠平滑肌细胞线粒体膜电位变化与细胞凋亡的关系。
Objective To investigate the relationship between mitochondrial membrane potential change and apoptosis of smooth muscle cells of the small intestine in diabetic rats.
心脏是血液循环的动力源，心肌细胞线粒体在此过程为细胞活动提供能量。
Heart is the power source of hemocircular, and the mitochondria of cadiocyte provided energy for cytolergy in this process.
目的探讨线粒体脑肌病患者骨骼肌细胞线粒体DNA基因突变情况及发病机制。
Objective To study the gene mutation in mitochondrial DNA in the patients with mitochondrial encephalomyopathies, and the pathogenic mechanism of mitochondrial encephalomyopathies.
目的探讨风湿性心脏病心肌细胞线粒体膜磷脂损伤与换瓣术后低心排血量的关系。
Objective To explore the relationship between myocytic mitochondrial membrane phospholipid(mtMPL) injure of rheumatic heart disease and low cardiac output following cardiac valve replacement.
使心肌细胞水肿和线粒体肿胀减轻。
透射电镜观察显示，哇巴因组大鼠心肌细胞肿胀，肌纤维断裂，Z线消失，线粒体增生肿胀，部分空泡化，细胞间胶原纤维增生。
Myofibrillar fragmentation, swelling of the cardiac myocytes, absence of the Z line, increases of the mitochondria and collagen fibers were found in ouabain group by transmission electron microscopy.
实验目的：研究内关穴位注射香丹注射液预处理对大鼠心肌缺血-再灌注损伤心肌细胞凋亡线粒体途径的影响。
Objective:To study the effect of pretreatment for mitochondrial pathway of ischemia-reperfusion (I/R) by injecting Xiangdan Zhusheye into PC6.
本文报道了小白鼠胚胎及生后在心肌细胞分化过程中线粒体的形态、分布和数量的变化。
This paper deals with, during myocardium differentiation, the morphology of mitochondrion and variations of distribution and quantity in embryo and after birth of mice.
图2缺氧再灌注无铈组：心肌细胞水肿，线粒体肿胀，嵴模糊。
Fig. 2 Anoxia reperfusion group without the cerium ions: Myocardial cell appears edema, mitochondria swell, cristae vague.
目的探讨大鼠冠状动脉微栓塞后心肌细胞凋亡及其线粒体途径激活的动态变化规律。
Objective to research the dynamic changes of cardiomyocyte apoptosis and the mechanism of mitochondria apoptosis pathway in rats after coronary microembolization.
结论小鼠及大鼠一次性急性力竭运动可致心肌细胞水肿与线粒体损伤。
Conclusion Acute exhaustive exercise can induce myocardial edema and mitochondrial damage in mice and rats.
图4缺氧再灌注加铈组：心肌细胞内线粒体肿胀，嵴模糊不清。
Fig. 4 Anoxia reperfusion group with the addition cerium ions: Mitochondria swelling and cristae vague appear in the cell.
图4缺氧再灌注加铈组：心肌细胞内线粒体肿胀，嵴模糊不清。
Fig. 4 Anoxia reperfusion group with the addition cerium ions: Mitochondria swelling and cristae vague appear in the cell.
应用推荐