目的通过慢性铝中毒造成老年痴呆小鼠模型，探讨其对M受体含量的影响。
Objective To bring about model mice with Alzheimer′s disease(AD)caused by aluminum chloride and to discuss effect on content of M receptor.
阿里达什使用小鼠水迷宫，来评估他的老年痴呆症小鼠的空间记忆。
Arendash USES a mouse maze to assess the spatial memory of his Alzheimer's mice.
结论：脑尔康能明显降低老年痴呆模型小鼠皮层及海马胆碱酯酶活性，并有保护海马ca 1区神经元的作用。
CONCLUSION: Naoerkang is able to decrease the acetylcholinesterase activity in the cerebral cortex and hippocampus of AD model mice and protect the neurons in the hippocampal CA1 region.
进一步的研究证明，当归益智方能降低痴呆小鼠脑的过氧化脂质水平和B型单胶氧化酶的活性，说明当归益智方对治疗老年痴呆有一定的作用。
The further studies indicated that it could depress the lipid peroxide level and B-type monoamine oxidase activity of dementia mice. which suggested that it have a certain action on senile dementia.
进一步的研究证明，当归益智方能降低痴呆小鼠脑的过氧化脂质水平和B型单胶氧化酶的活性，说明当归益智方对治疗老年痴呆有一定的作用。
The further studies indicated that it could depress the lipid peroxide level and B-type monoamine oxidase activity of dementia mice. which suggested that it have a certain action on senile dementia.
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