目的：探讨早期触摸和环境刺激对宫内缺血缺氧性脑损伤(HIBD)大鼠脑功能的影响。
Objective: To study the effect of the neonatal handling and enriched environmental stimulation on brain function of rats with hypoxic-ischemic brain damage (HIBD) in uterus.
肝组织均未发生显著变化。结论宫内窘迫时，脑组织首先出现自由基变化，为新生儿缺血缺氧性脑损伤的早期防治提供了理论依据。
Conclusion After severe intrauterine fetal distress, changes of free radicals occurred in brains firstly, which provides the proof of early treatment for neonatal hypoxia ischemia brain damage.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
目的探讨内源性一氧化碳（CO）在新生大鼠缺氧缺血性脑损伤（HIBD）中的作用机制及锡原卟啉的保护作用。
Objective To study the role of endogenous carbon monoxide(CO) in newborn rats with hypoxic-ischemic brain damage (HIBD) and the protective effect of Sn-protoporphyrin (SnPP).
目的：观察新生大鼠缺氧缺血性脑损伤(HIBD)后海马区细胞增殖的情况，探讨脑组织内源性修复的可能机制。
Objective to observe the expression of proliferation in hippocampus following hypoxic-ischemic brain damage (HIBD) in neonatal rats and to explore the possible endogenous neurogenesis of brain.
结论：新生大鼠缺氧缺血性脑损伤神经元退行性变形式多样。
Conclusion: This neuron degeneration of HIE in neonatal rats displayed a morphological diversity.
该文主要阐述细胞凋亡与新生儿缺氧缺血性脑损伤的关系，提出细胞凋亡机制在缺氧缺血性脑损伤中的重要性。
In this review, we summarized relationship between apoptosis and neonatal hypoxic-ischemic brain damage (HIBD) and importance of apoptosis mechanism in HIBD.
该文主要阐述细胞凋亡与新生儿缺氧缺血性脑损伤的关系，提出细胞凋亡机制在缺氧缺血性脑损伤中的重要性。
In this review, we summarized relationship between apoptosis and neonatal hypoxic-ischemic brain damage (HIBD) and importance of apoptosis mechanism in HIBD.
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