目的观察缺氧缺血性脑损伤新生猪脑电生理的变化。
Objective To observe the electric physiologic changes in newborn piglets with HIBD.
现将针刺对缺血性脑损伤作用机制的研究现状综述如下。
In this article, we have reviewed the mechanism of acupuncture on ischemic injury of brain.
目的：观察大鼠缺血性脑损伤一氧化氮(NO)含量变化。
Objective: To observe the change of the NO content in mices ischemic cerebral trauma.
结论：新生大鼠缺氧缺血性脑损伤神经元退行性变形式多样。
Conclusion: This neuron degeneration of HIE in neonatal rats displayed a morphological diversity.
目的研究山茶花总黄酮(TFC)对缺血性脑损伤的保护作用。
Objective to observe the protective effect of total flavone of Camellia (TFC) against cerebral ischemic injury.
氧化应激与缺血性脑损伤有关，氧化剂在脑损伤中起到重要作用。
Oxidative stress has been implicated in brain injury after ischemia, and the oxidants play an important role in cerebral damage.
目的：观察缺血性脑损伤对内源性神经干细胞增殖、迁移的影响。
AIM: To observe influences of ischemic brain injury on proliferation and transference of endogenous neural stem cells.
然而新生儿缺氧缺血性脑损伤后，准确判断其临床预后是困难的。
However neonatal hypoxic-ischemic brain injury and accurately determine the clinical prognosis is difficult.
目的：探讨阿司匹林预处理对缺血性脑损伤的保护机制及最佳剂量。
Objective: To study the neuroprotective mechanism of aspirin preconditioning in ischemic cerebral injury and its optimal dose.
缺血性脑损伤的发生机制目前尚未明确，临床缺乏有效的治疗手段。
The mechanism of ischemic brain injury has not been elucidated thoroughly now, and there is no effective therapy in clinic.
结果纳入缺氧缺血性脑损伤患儿148例，全部进入结果分析，无脱落。
RESULTS: All the 148 cases of hypoxia-ischemia brain injury were involved in the result analysis.
目的探索东莨菪碱对新生儿缺氧缺血性脑损伤（HIBD）的治疗作用。
Objective To evaluate treatment response of scopolamine for hypoxic-ischaemic brain damage(HIBD) in neonates.
目的：探讨激活素a在新生大鼠缺氧缺血性脑损伤中的表达规律及其意义。
Objective To investigate the effects of EGB on hepatic fibrosis and expression of Activin a in rats with fibrosis.
结论：脑组织产生过量的NO和血浆NO的不足参与了缺血性脑损伤的过程。
Conclusions: Excessive NO produced in brain tissue and insufficient plasma NO participate the course of ischemic brain damage.
围产期窒息所致缺氧缺血性脑损伤(HIBD)是新生儿死亡和残疾的主要原因。
Hypoxia ischemic brain damage (HIBD), which resulted from neonatal asphyxia, is the main cause of death and disability in neonatal periods.
背景：中国传统医学针灸治疗缺血性脑损伤可提高脑的抗损伤能力并加快损伤修复。
BACKGROUND: Acupuncture in Chinese traditional medicine improves capacity of brain on resisting injury and accelerates injury repair in treatment of ischemic brain injury.
目的：观察神昌滴丸对大鼠缺血性脑损伤后血液流变学及一氧化氮(NO)的影响。
Objective: To observe the influence of pills, "Shen-Chang", on Blood Rheology and Nitric Oxide (NO) after Ischemia Brain Injury in rats.
目的研究头皮药物位点注射对缺氧缺血性脑损伤(HIBD)后脑细胞的修复作用。
Objective To study the repairing effects of epicranium medication injection treatment on brain cells of hypoxic-ischemic brain damage (HIBD) and its mechanism.
目的：探讨头部药物位点注射对缺氧缺血性脑损伤(HIBD)后学习和记忆的影响。
Aim: To study the effects of epicranium medication injection treatment on learning and memory in brain cells of hypoxic-ischemic brain damage (HIBD).
缺血性脑损伤后，活性氧自由基(ROS)增加，以不同的细胞分子机制引起组织损伤。
After brain injury by ischemia, the production of reactive oxygen species (ROS) may increase, leading to tissues damage via several different cellular molecular pathways.
目前应用NSC治疗缺血性脑损伤包括运用外源性nsc移植和内源性NSC两种方法。
At present, NSC used for treating cerebral ischemia including extrinsic NSC transplantation and endogenous NSC.
新型脑保护剂通过不同机制作用于缺血性脑损伤的多个环节，为临床治疗提供了新的思路。
New brain protective agents for ischemic injury act in many aspects, providing new concepts and methods in clinical treatment.
目的探讨早期干预对缺氧缺血性脑损伤(HIBD)大鼠学习记忆能力及神经元凋亡的影响。
Objective to explore the influences of early interventions on learning, memory and neuron apoptosis in the brain of neonatal rats with hypoxic ischemic brain damage (HIBD).
结论高压氧对缺血性脑损伤有显著治疗作用，提高脑红蛋白的表达水平可能是其治疗的机制之一。
Conclusion Hyperbaric oxygen treatment had an obvious effect on ischemic brain injury, and increasing the expression of Neuroglobin might be one of the possible mechanisms of the therapy.
目的观察缺氧缺血性脑损伤（HIBD）新生大鼠白细胞介素-16（IL-16）表达的变化。
Objective To observe the variations in the expression levels of interleukin-16(IL-16) protein in neonatal rats with hypoxic-ischemic brain damage(HIBD).
观察缺氧缺血性脑损伤(HIBD)新生大鼠长时间吸高浓度氧后脑病理学及DNA损伤的变化。
To explore the pathologic and DNA changes in the brain of the hypoxic ischemic brain damage (HIBD) newborn rats after exposed to high oxygen for long time.
电针可以增加大鼠脑缺血区皮层ngf的分泌和表达时程，这可能是其保护缺血性脑损伤的机制之一。
Ea can increase the secretion and expression period of NGF in rats ischemic cortex, and this may be one of its protective mechanisms for ischemic cerebral damage.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
目的：探讨尼莫地平对磷脂酶a 2激活致急性缺血性脑损伤中的保护作用。设计：完全随机对照实验。
OBJECTIVE: To discuss the protective effects of nimodipine on acute ischemic brain injury caused by activation of phospholipase A2. DESIGN: a completely randomized controlled trial.
结论：阿司匹林预处理对随后的缺血性脑损伤具有保护作用，其机制可能是抑制NOS活性，减少NO释放。
Conclusions: Aspirin preconditioning has neuroprotective effects for the subsequent ischemic cerebral injury. Its mechanism may be the inhibition of NOS activity and the reduction of NO release.
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