通过亚低温对新生鼠缺氧缺血性脑损伤（HIBD）大脑皮质神经元一氧化氮合酶（NOS）及血糖水平影响的研究，探讨亚低温对缺氧缺血性脑损伤的保护作用机制。
Through investigating the effect of mild hypothermia on activity of nitric oxide synthase(NOS) in cortical neurons and glycemia levels of neonatal rats with hypoxic ischemic brain damage(HIBD).
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
脑缺血可上调大脑皮层和海马的神经元再生，这可能是一种重要的神经保护机制。
Ischemia could stimulate neurogenesis in the cerebral cortex and hippocampus which is considered as a very important neuronal protection mechanism.
而星形胶质细胞的激活支持和保护大脑免受脑缺血损伤。
The activation of astrocytes help ischemic brain repair and prevent from injury.
电针可以通过提高BDNF在缺血区周围皮层的表达，保护缺血性脑损伤，并可能与大脑可塑性的形成有一定的关系。
Ea has a protective effect on ischemic brain injury by increasing the BDNF expression in cortex around the ischemic focus.
结论P CF对大鼠大脑中动脉缺血再灌注损伤具有保护作用。
Conclusion PCF has the protective effects on brain nerves after cerebral ischemia and reperfusion.
目的：观察大鼠大脑中动脉(MCA)缺血后MMP - 2及TIMP - 2的动态变化及开博通对局灶性脑缺血大鼠的脑保护作用。
Objective to observe the dynamic changes of MMP-2 and TIMP-2 after middle cerebral artery (MCA) ischemia in rats and evaluate the neuroprotective effects of Captopril on focal cerebral ischemia.
目的：观察大鼠大脑中动脉(MCA)缺血后MMP - 2及TIMP - 2的动态变化及开博通对局灶性脑缺血大鼠的脑保护作用。
Objective to observe the dynamic changes of MMP-2 and TIMP-2 after middle cerebral artery (MCA) ischemia in rats and evaluate the neuroprotective effects of Captopril on focal cerebral ischemia.
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