越来越多的基础研究证明吸入麻醉剂在缺血预适应和缺血后适应中都具有心肌保护作用；
Various laboratory studies have demonstrated that inhalation anesthetics play a protective role in the myocardial ischemic preconditioning and postconditioning.
结论缺血预适应可升高缺血再灌注损伤后肾脏HSP70的表达，改善肾功能。
Conclusion Kidney ischemic preconditioning could increase the expression of HSP70 and decrease the injury of ischemia reprefusion.
缺血预适应是指心脏遭受短暂缺血后能耐受随后较长时间缺血损伤，是近年发现的一种预防心肌缺血损伤的有效措施。
Ischemic preconditioning has been defined as a tolerance of the myocardium to subsequent sustained ischemic damage after the heart was insulted one or more brief periods of ischemic stress.
目的探讨热处理、心肌缺血及缺血预适应后大鼠心脏、肝脏热休克蛋白(HSP70)的产生规律。
Objective To study the regular pattern of heat shock protein (HSP70) produced after heat shock, myocardial ischemia or ischemia preconditioning in heart and liver of rat.
目的评价缺血预适应(IPC)防治猪急性心肌梗死(ami)再灌注后无再流的作用。
Objective To evaluate the effects of ischemic preconditioning (IPC) on myocardial no - reflow in a mini-swine model of acute myocardial infarction (AMI) and reperfusion.
缺血预适应现象与再灌注后氧自由基生成增多、细胞内钙超载、细胞凋亡等有关。
It is often related with increasing oxygen free radical, calcium overload and cell apoptosis.
肢体缺血预适应是指肢体短暂缺血后使肢体本身骨骼肌或远隔组织能耐受较长时间的缺血损伤。
Ischemic preconditioning in limb renders skeletal muscle itself or remote tissue or organ being able to resistant a subsequent more sustained ischemic insult.
肢体缺血预适应是指肢体短暂缺血后使肢体本身骨骼肌或远隔组织能耐受较长时间的缺血损伤。
Ischemic preconditioning in limb renders skeletal muscle itself or remote tissue or organ being able to resistant a subsequent more sustained ischemic insult.
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