这是最常见的问题如凶险的败血症或慢性缺氧损害或心脏衰竭。
This is most commonly a problem in overwhelming sepsis or in the chronically hypoxically stressed or failing myocardium.
目的探讨新生儿缺氧缺血性脑病(HIE)围生因素与HIE分度、并发多器官损害以及电解质代谢紊乱的关系。
Objective to study the perinatal factors of hypoxia and ischemic encephalopathy (HIE) and the relationship between HIE and its grading, complicated multiple organ injury and electrolyte disorder.
目的探讨声学密度定量（AD）技术定量评价缺氧肺动脉高压（PH）后左室心肌损害的应用价值。
Objective To evaluate the value of acoustic densitometry (AD) in detecting myocardium damage of the left ventricle secondry to hypoxic pulmonary hypertension (PH).
背景：脑损伤后常引起继发性脑缺血、缺氧，使脑损害加重。
BACKGROUND: brain injury often causes secondary cerebral ischemia and hypoxia, which aggravate the brain damages.
缺氧及肺动脉高压导致血管内皮细胞损害，促进ET - 1释放是其重要因素。
Hypoxia and pulmonary hypertension could damage vascular endothelial cells and caused the release of ET-1.
目的：探讨1,6 -二磷酸果糖(FDP)对新生儿窒息后缺氧缺血性心肌损害的疗效。
Objective: to investigate the effect of fructose-1, 6-diphosphate (FDP) to treat hypoxic ischemic myocardial damage due to neonatal asphyxia.
结论CK、CK - MB值可作为新生儿缺氧缺血性心肌损害早期诊断的特异性指标，FDP可用于其治疗。
Conclusion CK and CK-MB can be used as the special forepart diagnose specifications of neonatal hypoxic-ischemic myocardial lesion, and FDP is one of the effective treatments.
目的：研究缺氧缺血性脑损害新生猪海马皮层病理改变及ATP生成率、ATP合成酶活性变化。
Objective: To study the changes of pathology, ATP production rate and ATP synthase activity in hippocampal cortex of neonatal pigs after hypoxic-ischemic brain damage (HIBD).
结果EEF可有效地改善腹腔脏器的缺血缺氧，并在此基础上减轻腹腔脏器的功能损害。
Results EEF could effectively improve the ischemic and insufficient oxygenation stage of splanchnic organs, and therefore alleviate the functional damage of abdominal viscera.
目的探讨新生儿缺氧缺血性脑病(HIE)与消化道损害的关系。
Objective To study the relationship between digestive tract damage and neonatal hypoxic-ischemic encephalopathy (HIE).
目的研究氟桂利嗪(FNZ)、拉莫三嗪(ltg)及两药联合经孕鼠给药，对胎鼠宫内缺血缺氧性脑损害的保护作用。
Objective To investigate the neuroprotective effects of flunarizine (FNZ) and lamotrigine (LTG), and their combination on the hypoxic-ischemic brain damage in fetal rats.
如果缺血和缺氧是严重的或长期的，心肌细胞就会受到严重的毁灭性的损害和坏死。
If the blood and oxygen supply is cut off severely or for a long period of time, muscle cells of the heart suffer severe and devastating damage and die.
结论：先心病不延迟婴儿期脑干发育，缺氧发作可损害脑干功能，对缺氧发作者应尽早进行干预。
Conclusion: CHD do not delay the development of brainstem, but ischemia attacks may harm brainstem function, so early intervention is needed for ischemia attack patients.
结论音频电预防性治疗可改善由低压缺氧造成的记忆功能损害，并对急性缺氧所致的中枢抑制有改善作用。
Conclusion Preventive therapy of MET can improve learning and memory impairment and also improve central inhibition induced by hypobaric hypoxia.
结论急性低压缺氧可引起老龄大鼠大脑皮质和丘脑中et、NO含量明显升高以及大脑皮质血管内皮细胞和神经细胞明显损伤，ET、NO可能参与了缺氧性脑损害的病理过程。
Conclusion It is suggested that, the increase of ET and NO contents of cerebral cortex and thalamus may participate in the pathogenesis of hypoxia brain injury in old rats.
本文主要介绍高级中枢、缺氧后脑血流量变化、缺氧对呼吸中枢神经元的实质性损害、神经递质和神经调质在缺氧性呼吸抑制过程中的作用。
This article introduces the effect of high central nervous system, changes of cerebral blood flow after hypoxia, damage of hypoxia to neurons in the respiratory center, neurotransmitters a…
目的探讨新生儿缺氧缺血性心肌损害临床表现及1,6 -二磷酸果糖(FDP)治疗疗效。
Objective to study the clinical feature of neonatal hypoxic-ischemic myocardial lesion and the efficacy of Fructose-1, 6-diphosphate (FDP).
目的探讨新生儿缺氧缺血性心肌损害临床表现及1,6 -二磷酸果糖(FDP)治疗疗效。
Objective to study the clinical feature of neonatal hypoxic-ischemic myocardial lesion and the efficacy of Fructose-1, 6-diphosphate (FDP).
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