目的探讨缺氧性脑病的治疗。
Objective To investigate the treatment of the anoxia brain disease.
方法回顾9例缺氧性脑病的治疗方案及治疗结果。
Methods To retrospective analyze the ways and the results of the 9 cases with the patients anoxia brain disease.
目的：研究缺氧性肺动脉高压大鼠右心室重构情况。
Objective: To study the pattern of right ventricle remodeling in hypoxic pulmonary hypertension rats.
结论米非司酮可造成中期妊娠脐静脉的缺氧性损伤。
Conclusion Mifepristone can cause a hypoxic damage in the second trimester fetal umbilical vein.
新生儿期发现缺血缺氧性脑病1例，颅内出血4例。
In neonatal stadium, 1 patient was found with hypoxia-ischemic encephalopathy, and 4 with cerebral hemorrhage.
目的：探讨纳洛酮对新生儿缺血缺氧性脑病的治疗作用。
Objective:To investigate the therapeutic effect of naloxone on ischemic hypoxic cerebropathy in neonates.
目的：观察肺心汤对慢性缺氧性肺动脉高压大鼠的作用。
Objective: To observe effects of Feixin Tang (FXT) on the chronic hypoxia pulmonary hypertension rats.
目的探讨一氧化氮对慢性缺氧性肺动脉高压的调节作用。
Objective the aim of this study was to examine the modulatory role of endogenous nitric oxide in the development of chronically hypoxic pulmonary hypertension.
目的探讨高压氧疗法对缺血缺氧性脑病(HIE)的疗效。
Objective To discuss the curative effect of HIE treated with HBO.
目的探讨慢性缺氧引起缺氧性肺血管收缩反应降低的机制。
Objective To investigate the mechanism of chronic hypoxia induced decreased HPV.
结论：缺氧性肺动脉高压大鼠右心室早期表现为离心性肥大。
Conclusion : Eccentric hypertrophy accounts for the early change of right ventricle in hypoxic pulmonary hypertension rats.
目的探讨超声在新生儿缺血缺氧性脑病(HIE)的诊断价值。
Objective Explore the ultrasound in the neonatal hypoxic ischemic encephalopathy (HIE) the diagnostic value.
认为主要的致死因素是神经细胞的缺氧性坏死和脑内弥漫性出血。
The main factors causation death of the animal were anoxemic necrosis of neurons and diffuse intracerebral hemorrhage.
目的观察新生鼠缺血缺氧性损伤后皮层及海马神经干细胞的变化。
Objective To investigate the influence of hypoxic- ischemic(H-I) injury on the endogenous Neural Stem Cells (NSCs) of the cerebral cortex and hippocampus in the neonatal rats.
目的探讨围产期缺氧性脑损伤患儿神经系统后遗症的早期预测方法。
Objective To find out ways of early prediction of the prognosis of perinatal hypoxic brain damage.
目的：探讨磁场对缺氧性心肌损伤大鼠血浆MDA含量变化的影响。
Objective To study the effects of magnetic field on the content of serum MDA in the rats with ischemic myocardial damage.
方法：将8头平原幼猪空运到高原，形成急性缺氧性肺动脉高压模型。
Methods: 8 piglets were transported to high altitude areas from plain by plane and the acute pulmonary hypertension model was established.
目标：确定窒息新生儿罹患新生儿缺血缺氧性脑病(HIE)的危险因素。
OBJECTIVE: to determine the risk factors for hypoxic-ischemic encephalopathy (HIE) in asphyxiated newborn infants.
以上结果表明，红细胞增多在缺氧性肺动脉高压和右室肥大中起着重要作用。
The above results proved that polycythemia might play an important role in pathogenesis of hypoxic pulmonary hypertension and myocardial hypertrophy.
结论一氧化碳对缺氧性肺血管结构重建大鼠肺动脉中TGF表达有抑制作用。
Conclusion Carbon monoxide inhibited TGF expression by pulmonary arteries of rats with hypoxic pulmonary vascular remodeling.
结论：螺旋CT头颅检查对新生儿脑缺血缺氧性脑病的临床分度有重要意义。
Conclusion:Sprial CT is important in the detection of neonatal hypoxic_ischemic encephalopathy.
结论：纳洛酮在一定程度上能改善新生儿缺血缺氧性脑病的病情，促进其恢复。
Conclusion: Naloxone can improve the conditions of ischemic hypoxic cerebropathy in neonates to a definite degree and facilitates recovery.
目的应用多模式诱发电位评估缺氧性脑病脑功能损伤程度和预测预后的准确性。
Objective To evaluate the brain function and predict the prognosis of hypoxic encephalopathy with multimodal evoked potentials.
结论一氧化氮对缺氧性肺血管结构重建大鼠肺动脉中PDGF表达有抑制作用。
Conclusion Nitric oxide inhibited PDGF expression by pulmonary arteries of rats with hypoxic pulmonary vascular remodeling.
目的研究吸入一氧化碳(CO)对急性缺氧性肺动脉高压及缺氧性肺损伤的影响。
Objective To investigate the effect of carbon monoxide (CO) inhalation on acute hypoxic pulmonary arterial hypertension and acute hypoxic lung injury.
目的：研究水蛭提取物对体外缺氧性新生大鼠大脑皮层神经细胞凋亡的保护作用。
Objective:To study the protective effect of leech extract on primary cultured neonate rat cerebral cortical neurons from the apoptosis caused by hypoxia in vitro.
目的探讨红花黄色素(SY)缓解大鼠心肌缺氧性损伤作用及其对能量代谢的影响。
Object To investigate the action of safflower yellow (SY) against rat cardiac hypoxic damage and the effect on energy metabolism.
目的探讨肺动脉局部肾素-血管紧张素系统对缺氧性肺动脉高压(PAH)的影响。
Objective To study the effect of local pulmonary arterial renin angiotensin system on hypoxic pulmonary hypertension (PAH).
以上结果表明红细胞增多可能在慢性缺氧性肺动脉高压和右室肥大发生起着重要作用。
These results proved that polycythemia may play an important role in pathogenesis of chronic hypoxic pulmonary hypertension and cardiac hypertrophy.
以上结果表明红细胞增多可能在慢性缺氧性肺动脉高压和右室肥大发生起着重要作用。
These results proved that polycythemia may play an important role in pathogenesis of chronic hypoxic pulmonary hypertension and cardiac hypertrophy.
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