血管内皮细胞分泌的强缩血管物质。
Vascular endothelial cell excretes , strong vasoconstrictor.
结论：高血压时血管细胞为抵消或减轻et -1等缩血管物质的过度分泌而代偿性的合成和分泌adm增多。
Conclusion: During the course of hypertension, vascular cell synthesize and secrete more ADM to compensate for the excessive secretion of et 1.
提示MT可以通过抑制ET的释放和拮抗ET的缩血管效应，发挥其内源性抗损伤物质的作用。
Tihe resulis suggest that MT could exert its injury-resistant action, as an endogenous defensive substance through inhibition of ET release and anlagonization against ET-induced vasocontraction.
提示MT可以通过抑制ET的释放和拮抗ET的缩血管效应，发挥其内源性抗损伤物质的作用。
Tihe resulis suggest that MT could exert its injury-resistant action, as an endogenous defensive substance through inhibition of ET release and anlagonization against ET-induced vasocontraction.
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